Nrf2 deficiency aggravates the increase in osteoclastogenesis and bone loss induced by inorganic arsenic.


Journal

Toxicology and applied pharmacology
ISSN: 1096-0333
Titre abrégé: Toxicol Appl Pharmacol
Pays: United States
ID NLM: 0416575

Informations de publication

Date de publication:
15 03 2019
Historique:
received: 28 12 2018
revised: 06 02 2019
accepted: 07 02 2019
pubmed: 12 2 2019
medline: 20 12 2019
entrez: 12 2 2019
Statut: ppublish

Résumé

Arsenic exposure increases the risk of various bone disorders. For instance, chronic exposure to low level arsenic can cause bone resorption by promoting osteoclast differentiation. Osteoclast precursor cells produce hydrogen peroxide after low level arsenic exposure and then undergo differentiation, producing cells which break down bone matrix. Nuclear factor E2-related factor 2 (Nrf2) regulates receptor activator of nuclear factor-κB dependent osteoclastogenesis by modulating intracellular reactive oxygen species (ROS) signaling via expression of cytoprotective enzymes. Here we tested the hypothesis that loss of Nrf2 will increase arsenic-induced bone loss. We treated 40 week-old Nrf2

Identifiants

pubmed: 30742845
pii: S0041-008X(19)30052-3
doi: 10.1016/j.taap.2019.02.003
pii:
doi:

Substances chimiques

Arsenites 0
NF-E2-Related Factor 2 0
Nfe2l2 protein, mouse 0
Sodium Compounds 0
sodium arsenite 48OVY2OC72

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

62-70

Commentaires et corrections

Type : ErratumIn

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Zhiyuan Liu (Z)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Yongyong Hou (Y)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Lu Li (L)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Yang Yang (Y)

Department of Rehabilitation, The First Affiliated Hospital of China Medical University, No. 155 Nanjing North Road, Heping Area, Shenyang 110001, China.

Jingkun Jia (J)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Zhixuan Hong (Z)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Tingfeng Li (T)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Yuanyuan Xu (Y)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Jingqi Fu (J)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China.

Yongxin Sun (Y)

Department of Rehabilitation, The First Affiliated Hospital of China Medical University, No. 155 Nanjing North Road, Heping Area, Shenyang 110001, China.

Masayuki Yamamoto (M)

Department of Medical Biochemistry, Tohoku University Graduate School of Medicine, Sendai 980-8575, Japan.

Huihui Wang (H)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China. Electronic address: hhwang@cmu.edu.cn.

Jingbo Pi (J)

School of Public Health, China Medical University, No. 77 Puhe Road, Shenyang North New Area, Shenyang 110122, China. Electronic address: jbpi@cmu.edu.cn.

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Classifications MeSH