Ouabain promotes immune responses in WEHI-3 cells to generate leukemia mice through enhancing phagocytosis and natural killer cell activities in vivo.


Journal

Environmental toxicology
ISSN: 1522-7278
Titre abrégé: Environ Toxicol
Pays: United States
ID NLM: 100885357

Informations de publication

Date de publication:
May 2019
Historique:
received: 05 12 2018
revised: 22 01 2019
accepted: 27 01 2019
pubmed: 15 2 2019
medline: 21 5 2019
entrez: 15 2 2019
Statut: ppublish

Résumé

Ouabain, a cardiotonic steroid, was used for the treatment of heart failure and atrial fibrillation and induces cancer cell apoptosis in many human cancer cells including human leukemia cells. However, there are no reports to show the effects on immune responses in a leukemia mouse model. In this study, WEHI-3 cell generated leukemia mice were developed and treated by oral ouabain at 0, 0.75, 1.5, and 3 mg/kg for 15 days. Results indicated that ouabain did not affect body appearance, but decreased liver and spleen weights, B- and T-cell proliferation at all three doses treatment and increased CD19 cells at 3.0 mg/kg treatment, decreased CD3, CD11b, and Mac-3 cells levels compared with positive control. Furthermore, ouabain increased the macrophage phagocytosis from peripheral blood mononuclear cell and peritoneal cavity at all three doses treatment and increased NK cell activities. Ouabain restored GOT, GPT and LDH levels in WEHI-3 leukemia mice in vivo.

Identifiants

pubmed: 30761740
doi: 10.1002/tox.22732
doi:

Substances chimiques

Antineoplastic Agents, Phytogenic 0
Ouabain 5ACL011P69

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

659-665

Subventions

Organisme : Experiments and data analysis were performed in part through the use of the Medical Research Core Facilities Center, Office of Research and Development at China Medical University, Taichung, Taiwan
Organisme : Shin Kong Wu Ho-Su Memorial Hospital
ID : SKH-8302-104-NDR-12
Organisme : Cheng Hsin General Hospital
ID : 104-38

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Auteurs

Yung-Luen Shih (YL)

Department of Pathology and Laboratory Medicine, Shin Kong Wu Ho-Su Memorial Hospital, Taipei, Taiwan.
School of Medical Laboratory Science and Biotechnology, Taipei Medical University, Taipei, Taiwan.
School of Medicine, College of Medicine, Fu-Jen Catholic University, New Taipei, Taiwan.

Hung-Sheng Shang (HS)

Graduate Institute of Clinical of Medicine, College of Medicine, Taipei Medical University, Taipei, Taiwan.
Division of Clinical Pathology, Department of Pathology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan.

Yung-Liang Chen (YL)

Department of Medical Laboratory Science and Biotechnology, Yuanpei University, Hsinchu, Taiwan.

Shu-Ching Hsueh (SC)

Division of Hematology and Oncology, Cheng Hsin General Hospital, Taipei, Taiwan.
Department of Family Medicine and Community Medicine, Cheng Hsin General Hospital, Taipei, Taiwan.

Hsiao-Min Chou (HM)

Department of Pathology and Laboratory Medicine, Shin Kong Wu Ho-Su Memorial Hospital, Taipei, Taiwan.

Hsu-Feng Lu (HF)

Department of Restaurant, Hotel and Institutional Management, Fu-Jen Catholic University, New Taipei, Taiwan.
Department of Clinical Pathology, Cheng Hsin General Hospital, Taipei, Taiwan.

Ming-Zhe Lee (MZ)

Department of Clinical Pathology, Cheng Hsin General Hospital, Taipei, Taiwan.

Hsin-Tu Hou (HT)

Department of Clinical Pathology, Cheng Hsin General Hospital, Taipei, Taiwan.

Ying-Ying Chuang (YY)

Department of Clinical Pathology, Cheng Hsin General Hospital, Taipei, Taiwan.

Mei-Hui Lee (MH)

Department of Genetic Counseling Center, Changhua Christian Hospital, Changhua, Taiwan.

Kuo-Wei Chen (KW)

Division of Hematology and Oncology, Cheng Hsin General Hospital, Taipei, Taiwan.

Jing-Gung Chung (JG)

Department of Biological Science and Technology, China Medical University, Taichung, Taiwan.
Department of Biotechnology, Asia University, Taichung, Taiwan.

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Classifications MeSH