Staphylococcus aureus TarP: A Brick in the Wall or Rosetta Stone?


Journal

Cell host & microbe
ISSN: 1934-6069
Titre abrégé: Cell Host Microbe
Pays: United States
ID NLM: 101302316

Informations de publication

Date de publication:
13 02 2019
Historique:
entrez: 15 2 2019
pubmed: 15 2 2019
medline: 14 5 2019
Statut: ppublish

Résumé

Staphylococcus aureus infection elicits antibodies against wall teichoic acid (WTA). Several glycosyltransferases modify WTA to generate anomeric heterogeneity. In recent work, Gerlach et al. (2018) show that modification by prophage-encoded TarP diminishes WTA immunogenicity, allowing staphylococci to evade host adaptive immune responses, and propose to exploit these insights for vaccines.

Identifiants

pubmed: 30763532
pii: S1931-3128(19)30051-4
doi: 10.1016/j.chom.2019.01.013
pmc: PMC6724547
mid: NIHMS1048075
pii:
doi:

Substances chimiques

Antibodies 0
Streptococcal Vaccines 0
Teichoic Acids 0
Glycosyltransferases EC 2.4.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

182-183

Subventions

Organisme : NIAID NIH HHS
ID : R01 AI110937
Pays : United States

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Références

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pubmed: 19868163
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pubmed: 23834243
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pubmed: 24045948
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pubmed: 26016486
Nature. 2015 Nov 19;527(7578):323-8
pubmed: 26536114
Nature. 2018 Nov;563(7733):705-709
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N Engl J Med. 1994 Apr 21;330(16):1107-13
pubmed: 8133853

Auteurs

Dominique Missiakas (D)

Department of Microbiology, The University of Chicago, 920 East 58(th) Street, Chicago, IL 60637, USA. Electronic address: dmissiak@bsd.uchicago.edu.

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Classifications MeSH