Impact on renal resistive index of diabetes in renal transplant donors and recipients: A retrospective analysis of 1827 kidney transplant recipients.


Journal

Journal of clinical hypertension (Greenwich, Conn.)
ISSN: 1751-7176
Titre abrégé: J Clin Hypertens (Greenwich)
Pays: United States
ID NLM: 100888554

Informations de publication

Date de publication:
03 2019
Historique:
received: 21 08 2018
revised: 19 10 2018
accepted: 30 10 2018
pubmed: 16 2 2019
medline: 25 6 2020
entrez: 16 2 2019
Statut: ppublish

Résumé

High renal resistive index (RI) is observed in diabetes and is associated with poor patient survival, but whether it is primarily due to renal vascular resistance or systemic vascular alterations is unclear. The respective impact of kidney transplant from diabetic donors or to diabetic recipients on RI would shed some light on this issue. The objective of the study was to analyze the impact of donor and recipient diabetes on RI in order to understand the respective impact of the kidney and the vascular environment. The authors conducted a retrospective study in 1827 renal transplant recipients who received a kidney between 1985 and 2017, and had Doppler measurements at 3 months after transplant. Donor and recipient characteristics at the time of transplant and at 3 months were reviewed. Both donor diabetes and recipient diabetes were associated with RI in univariate analysis, but only recipient diabetes remained significantly associated in stepwise multivariate analyses (effect estimate on RI: +0.03 ± 0.005, P < 0.001). These findings were confirmed when RI was expressed as a binary variable using a cutoff of 0.75 (OR = 2.50 [1.77, 3.54], P < 0.001). Other determinants of RI were recipient characteristics (age, sex, systolic and diastolic blood pressure, and duration of dialysis). Donor characteristics were not associated with RI. Our results suggest that high RI observed in diabetic recipients shortly after transplant is primarily due to the new vascular environment, rather than to characteristics of the transplanted kidney. Therefore, RI reflects systemic rather than intra-renal changes.

Identifiants

pubmed: 30767377
doi: 10.1111/jch.13492
pmc: PMC8030490
doi:

Substances chimiques

Creatinine AYI8EX34EU

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

382-389

Informations de copyright

©2019 Wiley Periodicals, Inc.

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Auteurs

Jean-Baptiste de Freminville (JB)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Louis-Marie Vernier (LM)

Néphrologie-Dialyses, Hopital Caremeau, Nimes, France.

Jérome Roumy (J)

Imagerie Médicale, Hôpital Bretonneau, CHRU Tours, Tours, France.

Frédéric Patat (F)

Imagerie Médicale, Hôpital Bretonneau, CHRU Tours, Tours, France.
CIC-IT 1415, CHRU Tours, Tours, France.

Philippe Gatault (P)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.
EA4245, François-Rabelais University, Tours, France.

Bénédicte Sautenet (B)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Elodie Bailly (E)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Eloi Chevallier (E)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Christelle Barbet (C)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Hélène Longuet (H)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Elodie Merieau (E)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Christophe Baron (C)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.
EA4245, François-Rabelais University, Tours, France.

Matthias Buchler (M)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.

Jean-Michel Halimi (JM)

Néphrologie-Immunologie Clinique, Hôpital Bretonneau, CHRU Tours, Tours, France.
EA4245, François-Rabelais University, Tours, France.

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