Dietary ascorbic acid restriction in GNL/SMP30-knockout mice unveils the role of ascorbic acid in regulation of somatic and visceral pain sensitivity.
Animals
Ascorbic Acid
/ metabolism
Ascorbic Acid Deficiency
/ complications
Calcium Channels, T-Type
/ metabolism
Calcium-Binding Proteins
/ genetics
Hyperalgesia
/ etiology
Intracellular Signaling Peptides and Proteins
/ genetics
Male
Mice, Inbred C57BL
Mice, Knockout
Pain Threshold
Visceral Pain
/ etiology
Ascorbic acid
Cav3.2 T-type calcium channel
Hydrogen sulfide
Neuropathy
Visceral pain
Vitamin C
Journal
Biochemical and biophysical research communications
ISSN: 1090-2104
Titre abrégé: Biochem Biophys Res Commun
Pays: United States
ID NLM: 0372516
Informations de publication
Date de publication:
09 04 2019
09 04 2019
Historique:
received:
12
02
2019
accepted:
20
02
2019
pubmed:
5
3
2019
medline:
22
1
2020
entrez:
5
3
2019
Statut:
ppublish
Résumé
Ca
Identifiants
pubmed: 30827506
pii: S0006-291X(19)30303-1
doi: 10.1016/j.bbrc.2019.02.102
pii:
doi:
Substances chimiques
Cacna1h protein, mouse
0
Calcium Channels, T-Type
0
Calcium-Binding Proteins
0
Intracellular Signaling Peptides and Proteins
0
Rgn protein, mouse
0
Ascorbic Acid
PQ6CK8PD0R
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
705-710Informations de copyright
Copyright © 2019 Elsevier Inc. All rights reserved.