Benign proliferative epithelial lesions of oral mucosa are infrequently associated with α-, β-, or γ human papillomaviruses.

HPV Oral benign p16 papilloma

Journal

Laryngoscope investigative otolaryngology
ISSN: 2378-8038
Titre abrégé: Laryngoscope Investig Otolaryngol
Pays: United States
ID NLM: 101684963

Informations de publication

Date de publication:
Feb 2019
Historique:
received: 29 03 2018
revised: 20 09 2018
accepted: 03 10 2018
entrez: 5 3 2019
pubmed: 5 3 2019
medline: 5 3 2019
Statut: epublish

Résumé

Oral papillomas and verruca vulgaris have been associated with human papillomavirus (HPV) infection. However, approximately half of these have remained HPV-negative when tested for mucosal HPV genotypes. In this study, we evaluated presence of α-, β-, and γ-HPVs in benign papillary and verrucous lesions. Eighty-three clinical lesions with suspected HPV etiology were analyzed for HPV types of genus α (n = 24), β (n = 46), and γ (n = 52). Immunohistochemistry was used for p16 as a possible surrogate marker of high-risk HPV, accompanied by Ki-67 proliferation marker. Altogether, α-HPVs were detected in 6.4%, β-HPVs in 2.4%, and γ-HPV in 4.8%. The following genotypes were identified: HPV6, 8, 11, 16, 22, 161, and 170. Neither Ki-67 nor p16 positivity alone were associated with HPV but combined staining showed significant inverse association ( HPV infection is found only in a minority of benign verrucous and papillary oral lesions, with the predominance of α-HPVs. 4.

Sections du résumé

BACKGROUND BACKGROUND
Oral papillomas and verruca vulgaris have been associated with human papillomavirus (HPV) infection. However, approximately half of these have remained HPV-negative when tested for mucosal HPV genotypes. In this study, we evaluated presence of α-, β-, and γ-HPVs in benign papillary and verrucous lesions.
METHODS METHODS
Eighty-three clinical lesions with suspected HPV etiology were analyzed for HPV types of genus α (n = 24), β (n = 46), and γ (n = 52). Immunohistochemistry was used for p16 as a possible surrogate marker of high-risk HPV, accompanied by Ki-67 proliferation marker.
RESULTS RESULTS
Altogether, α-HPVs were detected in 6.4%, β-HPVs in 2.4%, and γ-HPV in 4.8%. The following genotypes were identified: HPV6, 8, 11, 16, 22, 161, and 170. Neither Ki-67 nor p16 positivity alone were associated with HPV but combined staining showed significant inverse association (
CONCLUSION CONCLUSIONS
HPV infection is found only in a minority of benign verrucous and papillary oral lesions, with the predominance of α-HPVs.
LEVEL OF EVIDENCE METHODS
4.

Identifiants

pubmed: 30828618
doi: 10.1002/lio2.222
pii: LIO2222
pmc: PMC6383307
doi:

Types de publication

Journal Article

Langues

eng

Pagination

43-48

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Auteurs

Sirli Kerge (S)

Department of Oral Diseases Turku University Hospital Turku Finland.

Jessi Vuorinen (J)

Department of Oral Pathology University of Turku Turku Finland.

Saija Hurme (S)

Department of Biostatistics University of Turku Turku Finland.

Tero Soukka (T)

Department of Oral Diseases Turku University Hospital Turku Finland.

Tarik Gheit (T)

Infections and Cancer Biology Group International Agency for Research on Cancer Lyon France.

Massimo Tommasino (M)

Infections and Cancer Biology Group International Agency for Research on Cancer Lyon France.

Stina Syrjänen (S)

Department of Pathology Turku University Hospital Turku Finland.
Department of Oral Pathology University of Turku Turku Finland.

Jaana Rautava (J)

Department of Pathology Turku University Hospital Turku Finland.
Department of Oral Pathology University of Turku Turku Finland.

Classifications MeSH