miR-132 suppresses transcription of ribosomal proteins to promote protective Th1 immunity.
Animals
Binding Sites
CD4-Positive T-Lymphocytes
/ immunology
Cytokines
/ biosynthesis
Female
Gene Expression Profiling
Gene Expression Regulation
Gene Regulatory Networks
Host-Pathogen Interactions
/ genetics
Lymphocyte Activation
/ genetics
Mice
Mice, Transgenic
MicroRNAs
/ genetics
Protein Binding
RNA Interference
Ribosomal Proteins
/ genetics
Spleen
/ immunology
Th1 Cells
/ immunology
Transcription Factor TFIID
/ metabolism
p300-CBP Transcription Factors
/ metabolism
Leishmania
Th cells
miR‐132
microRNA
ribosomal proteins
Journal
EMBO reports
ISSN: 1469-3178
Titre abrégé: EMBO Rep
Pays: England
ID NLM: 100963049
Informations de publication
Date de publication:
04 2019
04 2019
Historique:
received:
20
06
2018
revised:
01
02
2019
accepted:
06
02
2019
pubmed:
6
3
2019
medline:
1
5
2020
entrez:
6
3
2019
Statut:
ppublish
Résumé
Determining the mechanisms that distinguish protective immunity from pathological chronic inflammation remains a fundamental challenge. miR-132 has been shown to play largely immunoregulatory roles in immunity; however, its role in CD4
Identifiants
pubmed: 30833344
pii: embr.201846620
doi: 10.15252/embr.201846620
pmc: PMC6446204
mid: EMS84321
pii:
doi:
Substances chimiques
Cytokines
0
MIRN132 microRNA, mouse
0
MicroRNAs
0
Ribosomal Proteins
0
Transcription Factor TFIID
0
p300-CBP Transcription Factors
EC 2.3.1.48
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Medical Research Council
ID : G1000230
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/L008505/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/N009185/1
Pays : United Kingdom
Informations de copyright
© 2019 The Authors.
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