Rapid, biphasic CRF neuronal responses encode positive and negative valence.


Journal

Nature neuroscience
ISSN: 1546-1726
Titre abrégé: Nat Neurosci
Pays: United States
ID NLM: 9809671

Informations de publication

Date de publication:
04 2019
Historique:
received: 10 04 2018
accepted: 18 01 2019
pubmed: 6 3 2019
medline: 22 5 2019
entrez: 6 3 2019
Statut: ppublish

Résumé

Corticotropin-releasing factor (CRF) that is released from the paraventricular nucleus (PVN) of the hypothalamus is essential for mediating stress response by activating the hypothalamic-pituitary-adrenal axis. CRF-releasing PVN neurons receive inputs from multiple brain regions that convey stressful events, but their neuronal dynamics on the timescale of behavior remain unknown. Here, our recordings of PVN CRF neuronal activity in freely behaving mice revealed that CRF neurons are activated immediately by a range of aversive stimuli. By contrast, CRF neuronal activity starts to drop within a second of exposure to appetitive stimuli. Optogenetic activation or inhibition of PVN CRF neurons was sufficient to induce a conditioned place aversion or preference, respectively. Furthermore, conditioned place aversion or preference induced by natural stimuli was significantly decreased by manipulating PVN CRF neuronal activity. Together, these findings suggest that the rapid, biphasic responses of PVN CRF neurons encode the positive and negative valences of stimuli.

Identifiants

pubmed: 30833699
doi: 10.1038/s41593-019-0342-2
pii: 10.1038/s41593-019-0342-2
pmc: PMC6668342
mid: NIHMS1519156
doi:

Substances chimiques

Corticotropin-Releasing Hormone 9015-71-8

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

576-585

Subventions

Organisme : NIDDK NIH HHS
ID : R01 DK106636
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH101377
Pays : United States

Commentaires et corrections

Type : CommentIn
Type : CommentIn

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Auteurs

Jineun Kim (J)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Seongju Lee (S)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Yi-Ya Fang (YY)

Neuroscience Institute, New York University School of Medicine, New York, NY, USA.
Department of Psychiatry, New York University School of Medicine, New York, NY, USA.

Anna Shin (A)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Seahyung Park (S)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Koichi Hashikawa (K)

Neuroscience Institute, New York University School of Medicine, New York, NY, USA.
Department of Psychiatry, New York University School of Medicine, New York, NY, USA.

Shreelatha Bhat (S)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Daesoo Kim (D)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Jong-Woo Sohn (JW)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea.

Dayu Lin (D)

Neuroscience Institute, New York University School of Medicine, New York, NY, USA. Dayu.Lin@nyulangone.org.
Department of Psychiatry, New York University School of Medicine, New York, NY, USA. Dayu.Lin@nyulangone.org.

Greg S B Suh (GSB)

Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Republic of Korea. Gregsuh@gmail.com.
Neuroscience Institute, New York University School of Medicine, New York, NY, USA. Gregsuh@gmail.com.
Skirball Institute of Biomolecular Medicine, New York University School of Medicine, New York, NY, USA. Gregsuh@gmail.com.
Department of Cell Biology, New York University School of Medicine, New York, NY, USA. Gregsuh@gmail.com.

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