T-cell and B-cell perturbations are similar in ART-naive HIV-1 and HIV-1/2 dually infected patients.


Journal

AIDS (London, England)
ISSN: 1473-5571
Titre abrégé: AIDS
Pays: England
ID NLM: 8710219

Informations de publication

Date de publication:
01 06 2019
Historique:
pubmed: 8 3 2019
medline: 22 7 2020
entrez: 8 3 2019
Statut: ppublish

Résumé

HIV-2 may slow progression of a subsequently acquired HIV-1 infection through cross-neutralizing antibodies and polyfunctional CD8 T cells. We hypothesized that HIV-1/2 dually infected patients compared with HIV-1-infected patients had more preserved immune maturation subsets and less immune activation of T and B cells. ART-naive patients with HIV-1 (n = 83) or HIV-1/2 dual (n = 27) infections were included in this cross-sectional study at an HIV clinic in Guinea-Bissau. Peripheral blood mononuclear cells (PBMCs) were analyzed by flow cytometry according to T-cell maturation and activation, regulatory T-cell fraction, and B-cell maturation and activation. HIV-1/2 dually infected patients had lower levels of HIV-1 RNA compared with patients with HIV-1 infection, but the levels of total HIV RNA (HIV-1 and HIV-2) were similar in the two patient groups. T-cell maturation, and proportions of regulatory T cells (FoxP3+) were also similar in the two groups. HIV-1/2 dually infected patients had higher proportions of CD4 and CD8 T cells positive for the activation marker CD38, but there was no difference in other T-cell activation markers (CD28, CTLA-4, PD-1). HIV-1/2 dually infected patients also had higher proportions of IgM-only B cells and plasmablasts. HIV-1/2 was not associated with less immune perturbations than for HIV-1 infection.

Sections du résumé

BACKGROUND
HIV-2 may slow progression of a subsequently acquired HIV-1 infection through cross-neutralizing antibodies and polyfunctional CD8 T cells. We hypothesized that HIV-1/2 dually infected patients compared with HIV-1-infected patients had more preserved immune maturation subsets and less immune activation of T and B cells.
METHODS
ART-naive patients with HIV-1 (n = 83) or HIV-1/2 dual (n = 27) infections were included in this cross-sectional study at an HIV clinic in Guinea-Bissau. Peripheral blood mononuclear cells (PBMCs) were analyzed by flow cytometry according to T-cell maturation and activation, regulatory T-cell fraction, and B-cell maturation and activation.
RESULTS
HIV-1/2 dually infected patients had lower levels of HIV-1 RNA compared with patients with HIV-1 infection, but the levels of total HIV RNA (HIV-1 and HIV-2) were similar in the two patient groups. T-cell maturation, and proportions of regulatory T cells (FoxP3+) were also similar in the two groups. HIV-1/2 dually infected patients had higher proportions of CD4 and CD8 T cells positive for the activation marker CD38, but there was no difference in other T-cell activation markers (CD28, CTLA-4, PD-1). HIV-1/2 dually infected patients also had higher proportions of IgM-only B cells and plasmablasts.
CONCLUSION
HIV-1/2 was not associated with less immune perturbations than for HIV-1 infection.

Identifiants

pubmed: 30845069
doi: 10.1097/QAD.0000000000002185
doi:

Substances chimiques

RNA, Viral 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1143-1153

Auteurs

Bo L Hønge (BL)

Bandim Health Project, Indepth Network, Bissau, Guinea-Bissau.
Department of Clinical Immunology.

Mikkel S Petersen (MS)

Department of Clinical Immunology.

Sanne Jespersen (S)

Bandim Health Project, Indepth Network, Bissau, Guinea-Bissau.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

Candida Medina (C)

National HIV Programme, Ministry of Health, Bissau, Guinea-Bissau.

David D S Té (DDS)

National HIV Programme, Ministry of Health, Bissau, Guinea-Bissau.

Bertram Kjerulff (B)

Department of Clinical Immunology.

Mads M Jensen (MM)

Bandim Health Project, Indepth Network, Bissau, Guinea-Bissau.
Department of Clinical Immunology.

Ditte Steiniche (D)

Bandim Health Project, Indepth Network, Bissau, Guinea-Bissau.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

Joakim Esbjörnsson (J)

Department of Laboratory Medicine, Lund University, Lund, Sweden.
Nuffield Department of Medicine, University of Oxford, Oxford, United Kingdom.

Alex L Laursen (AL)

Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

Christian Wejse (C)

Bandim Health Project, Indepth Network, Bissau, Guinea-Bissau.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.
GloHAU, Center for Global Health, School of Public Health, Aarhus University, Aarhus.

Henrik Krarup (H)

Section of Molecular Diagnostics, Clinical Biochemistry, Aalborg University Hospital, Denmark.

Bjarne K Møller (BK)

Department of Clinical Immunology.

Christian Erikstrup (C)

Department of Clinical Immunology.

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