Prognostic impact of TP53INP1 gene expression in estrogen receptor α-positive breast cancer patients.


Journal

Japanese journal of clinical oncology
ISSN: 1465-3621
Titre abrégé: Jpn J Clin Oncol
Pays: England
ID NLM: 0313225

Informations de publication

Date de publication:
01 Jun 2019
Historique:
received: 02 11 2018
revised: 01 02 2019
accepted: 13 02 2019
pubmed: 12 3 2019
medline: 17 7 2019
entrez: 12 3 2019
Statut: ppublish

Résumé

Tumor protein 53-induced nuclear protein 1 (TP53INP1) is a key stress protein with tumor suppressor function. Several studies have demonstrated TP53INP1 downregulation in many cancers. In this study, we investigated the correlations of TP53INP1 mRNA expression in breast cancer tissues with prognosis and the correlations of microRNAs that regulate TP53INP1 expression in breast cancer patients with long follow-up. A total of 453 invasive breast cancer tissues were analyzed for TP53INP1 mRNA expression. We examined correlations of clinicopathological factors and expression levels of TP53INP1 mRNA in these samples. The expressions of miR-155, miR-569 and markers associated with tumor-initiating capacity were also analyzed. The median follow-up period was 9.0 years. We found positive correlations between low expression of TP53INP1 mRNA and shorter disease-free survival and overall survival in breast cancer patients (P = 0.0002 and P < 0.0001, respectively), as well as in estrogen receptor α (ERα)-positive patients receiving adjuvant endocrine therapy (P = 0.01 and P = 0.0008, respectively). No correlations were found in ERα-negative patients. Low TP53INP1 mRNA levels positively correlated with higher grade and ERα-negativity. Multivariate analysis indicated that TP53INP1 mRNA level was an independent risk factor for overall survival both in overall breast cancer patients (hazard ratio, 2.13; 95% confidence interval, 1.17-3.92) and ERα-positive patients (hazard ratio, 2.34; 95% confidence interval, 1.18-4.64). We show that low expression of TP53INP1 is an independent factor of poor prognosis in breast cancer patients, especially ERα-positive patients. TP53INP1 might be a promising candidate biomarker and therapeutic target in ERα-positive breast cancer patients.

Sections du résumé

BACKGROUND BACKGROUND
Tumor protein 53-induced nuclear protein 1 (TP53INP1) is a key stress protein with tumor suppressor function. Several studies have demonstrated TP53INP1 downregulation in many cancers. In this study, we investigated the correlations of TP53INP1 mRNA expression in breast cancer tissues with prognosis and the correlations of microRNAs that regulate TP53INP1 expression in breast cancer patients with long follow-up.
METHODS METHODS
A total of 453 invasive breast cancer tissues were analyzed for TP53INP1 mRNA expression. We examined correlations of clinicopathological factors and expression levels of TP53INP1 mRNA in these samples. The expressions of miR-155, miR-569 and markers associated with tumor-initiating capacity were also analyzed. The median follow-up period was 9.0 years.
RESULTS RESULTS
We found positive correlations between low expression of TP53INP1 mRNA and shorter disease-free survival and overall survival in breast cancer patients (P = 0.0002 and P < 0.0001, respectively), as well as in estrogen receptor α (ERα)-positive patients receiving adjuvant endocrine therapy (P = 0.01 and P = 0.0008, respectively). No correlations were found in ERα-negative patients. Low TP53INP1 mRNA levels positively correlated with higher grade and ERα-negativity. Multivariate analysis indicated that TP53INP1 mRNA level was an independent risk factor for overall survival both in overall breast cancer patients (hazard ratio, 2.13; 95% confidence interval, 1.17-3.92) and ERα-positive patients (hazard ratio, 2.34; 95% confidence interval, 1.18-4.64).
CONCLUSIONS CONCLUSIONS
We show that low expression of TP53INP1 is an independent factor of poor prognosis in breast cancer patients, especially ERα-positive patients. TP53INP1 might be a promising candidate biomarker and therapeutic target in ERα-positive breast cancer patients.

Identifiants

pubmed: 30855679
pii: 5374485
doi: 10.1093/jjco/hyz029
doi:

Substances chimiques

Biomarkers, Tumor 0
Carrier Proteins 0
Estrogen Receptor alpha 0
Heat-Shock Proteins 0
MicroRNAs 0
TP53INP1 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

567-575

Informations de copyright

© The Author(s) 2019. Published by Oxford University Press. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Auteurs

Mayumi Nishimoto (M)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Sayaka Nishikawa (S)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Naoto Kondo (N)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Yumi Wanifuchi-Endo (Y)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Yukari Hato (Y)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Tomoka Hisada (T)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Yu Dong (Y)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Katsuhiro Okuda (K)

Department of Oncology, Immunology and Surgery, Nagoya City University Graduate School of Medical Sciences.

Hiroshi Sugiura (H)

Department of Education and Research Center for Advanced Medicine, Nagoya City University Graduate School of Medical Sciences.

Hiroyuki Kato (H)

Department of Experimental Pathology and Tumor Biology, Nagoya City University Graduate School of Medical Sciences, 1 Kawasumi, Mizuho-cho, Mizuho-ku, Nagoya, Japan.

Satoru Takahashi (S)

Department of Experimental Pathology and Tumor Biology, Nagoya City University Graduate School of Medical Sciences, 1 Kawasumi, Mizuho-cho, Mizuho-ku, Nagoya, Japan.

Tatsuya Toyama (T)

Department of Breast Surgery, Nagoya City University Graduate School of Medical Sciences.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH