Brief Report: Higher Levels of Angiopoietin-1 Are Associated With Early and Sustained Viral Suppression in Children Living With Vertically Acquired HIV.


Journal

Journal of acquired immune deficiency syndromes (1999)
ISSN: 1944-7884
Titre abrégé: J Acquir Immune Defic Syndr
Pays: United States
ID NLM: 100892005

Informations de publication

Date de publication:
15 04 2019
Historique:
entrez: 14 3 2019
pubmed: 14 3 2019
medline: 19 12 2019
Statut: ppublish

Résumé

Systemic inflammation, platelet dysfunction, and endothelial activation persist in people living with HIV despite sustained virologic suppression (SVS) with combined antiretroviral therapy (cART) and may lead to complications such as atherosclerosis and cardiovascular disease. Angiopoietin-1 (Ang-1) is a key regulator of angiogenesis and endothelial activation and has been studied as an objective biomarker in disease states such as atherosclerosis, sepsis, and severe malaria. Eight pediatric HIV care centers across Canada. Cross-sectional study of 61 children living with vertically acquired HIV on cART with undetectable RNA viral load. Plasma levels of Ang-1 were measured by ELISA and analyzed in relation to clinical characteristics abstracted from medical records. Ang-1 levels were directly correlated with clinical indices of virologic control: cumulative proportion of life on effective cART (ρ = +0.35, P = 0.0078) and cumulative proportion of life with SVS (ρ = +0.36, P = 0.0049). Furthermore, higher Ang-1 levels were associated with younger age at SVS (ρ = -0.56, P < 0.0001). These associations remained statistically significant in multivariable linear regression models adjusting for potential confounders (P < 0.05 for all associations). Early effective cART and SVS were associated with higher Ang-1 levels in children living with vertically acquired HIV-1.

Sections du résumé

BACKGROUND
Systemic inflammation, platelet dysfunction, and endothelial activation persist in people living with HIV despite sustained virologic suppression (SVS) with combined antiretroviral therapy (cART) and may lead to complications such as atherosclerosis and cardiovascular disease. Angiopoietin-1 (Ang-1) is a key regulator of angiogenesis and endothelial activation and has been studied as an objective biomarker in disease states such as atherosclerosis, sepsis, and severe malaria.
SETTING
Eight pediatric HIV care centers across Canada.
METHODS
Cross-sectional study of 61 children living with vertically acquired HIV on cART with undetectable RNA viral load. Plasma levels of Ang-1 were measured by ELISA and analyzed in relation to clinical characteristics abstracted from medical records.
RESULTS
Ang-1 levels were directly correlated with clinical indices of virologic control: cumulative proportion of life on effective cART (ρ = +0.35, P = 0.0078) and cumulative proportion of life with SVS (ρ = +0.36, P = 0.0049). Furthermore, higher Ang-1 levels were associated with younger age at SVS (ρ = -0.56, P < 0.0001). These associations remained statistically significant in multivariable linear regression models adjusting for potential confounders (P < 0.05 for all associations).
CONCLUSIONS
Early effective cART and SVS were associated with higher Ang-1 levels in children living with vertically acquired HIV-1.

Identifiants

pubmed: 30865052
doi: 10.1097/QAI.0000000000001955
pii: 00126334-201904150-00014
doi:

Substances chimiques

Angiopoietin-1 0
Anti-HIV Agents 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

590-595

Subventions

Organisme : CIHR
Pays : Canada

Auteurs

Vishrut Gulhati (V)

University of Alberta, Edmonton, Alberta, Canada.

Jeremy Soo (J)

University of Alberta, Edmonton, Alberta, Canada.

Doris G Ransy (DG)

Unité d'immunopathologie virale, Centre de recherche du CHU Sainte-Justine, Montréal, Québec, Canada.

Jason Brophy (J)

Children's Hospital of Eastern Ontario, Ottawa, Ontario, Canada.

Fatima Kakkar (F)

Faculté de médecine, Université de Montréal.

Ari Bitnun (A)

Department of Pediatrics, Hospital for Sick Children, University of Toronto.

Lindy Samson (L)

Children's Hospital of Eastern Ontario, Ottawa, Ontario, Canada.

Stanley Read (S)

Department of Pediatrics, Hospital for Sick Children, University of Toronto.

Hugo Soudeyns (H)

Unité d'immunopathologie virale, Department of Microbiology, Infectiology and Immunology, Centre de recherche du CHU Sainte-Justine, Université de Montréal.

Michael T Hawkes (MT)

Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.

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