SWI/SNF-Compromised Cancers Are Susceptible to Bromodomain Inhibitors.


Journal

Cancer research
ISSN: 1538-7445
Titre abrégé: Cancer Res
Pays: United States
ID NLM: 2984705R

Informations de publication

Date de publication:
15 05 2019
Historique:
received: 18 05 2018
revised: 18 10 2018
accepted: 12 03 2019
pubmed: 17 3 2019
medline: 15 2 2020
entrez: 17 3 2019
Statut: ppublish

Résumé

The antitumor activity of bromodomain and extraterminal motif protein inhibitors (BETi) has been demonstrated across numerous types of cancer. As such, these inhibitors are currently undergoing widespread clinical evaluation. However, predictive biomarkers allowing the stratification of tumors into responders and nonresponders to BETi are lacking. Here, we showed significant antiproliferative effects of low dosage BETi

Identifiants

pubmed: 30877105
pii: 0008-5472.CAN-18-1545
doi: 10.1158/0008-5472.CAN-18-1545
doi:

Substances chimiques

Biomarkers, Tumor 0
Nuclear Proteins 0
SMARCA2 protein, human 0
Transcription Factors 0
SMARCA4 protein, human EC 3.6.1.-
DNA Helicases EC 3.6.4.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

2761-2774

Subventions

Organisme : CIHR
ID : 399061
Pays : Canada
Organisme : CIHR
ID : 399114
Pays : Canada

Informations de copyright

©2019 American Association for Cancer Research.

Auteurs

Tatiana Shorstova (T)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Maud Marques (M)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Jie Su (J)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Jake Johnston (J)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Claudia L Kleinman (CL)

Department of Human Genetics, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Nancy Hamel (N)

Departments of Oncology and Human Genetics, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

Sidong Huang (S)

Department of Biochemistry, Rosalind and Morris Goodman Cancer Centre, McGill University, Montreal, Quebec, Canada.

Moulay A Alaoui-Jamali (MA)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada.

William D Foulkes (WD)

Departments of Oncology and Human Genetics, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada michael.witcher@mcgill.ca william.foulkes@mcgill.ca.

Michael Witcher (M)

Departments of Oncology and Experimental Medicine, McGill University, Lady Davis Institute and Segal Cancer Centre, Jewish General Hospital, Montreal, Quebec, Canada. michael.witcher@mcgill.ca william.foulkes@mcgill.ca.

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Classifications MeSH