Spectrum of Pulmonary Aspergillosis in Hyper-IgE Syndrome with Autosomal-Dominant STAT3 Deficiency.


Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
Historique:
received: 09 10 2018
revised: 26 02 2019
accepted: 28 02 2019
pubmed: 18 3 2019
medline: 25 9 2020
entrez: 18 3 2019
Statut: ppublish

Résumé

Autosomal-dominant signal transducer and activator of transcription 3 (STAT3) deficiency predisposes to recurrent bacterial pneumonia, complicated by bronchiectasis and cavitations. Aspergillosis is a major cause of morbidity in these patients. However, its diagnosis, classification, and treatment are challenging. We aimed to assess the prevalence and describe the clinical, mycological, and radiological presentation and related therapy and outcome of Aspergillus infections of the respiratory tract in the STAT3-deficient patients of the National French cohort. We performed a retrospective study of all pulmonary aspergillosis cases in STAT3-deficient patients (n = 74). Clinical and mycological data were collected up to October 2015 and imaging was centralized. Twenty-one episodes of pulmonary aspergillosis in 13 (17.5%) STAT3-deficient patients were identified. The median age at first episode was 13 years (interquartile range, 10-26 years). Ninety percent of patients had previous bronchiectasis or cavitations. Infections were classified as follows: 5 single aspergilloma, 9 chronic cavity pulmonary aspergillosis, 5 allergic bronchopulmonary aspergillosis-like disease, and 2 mixed forms of concomitant allergic bronchopulmonary aspergillosis-like disease and chronic cavity pulmonary aspergillosis. No invasive aspergillosis cases were identified. Aspergillus species were isolated in 71% of episodes and anti-Aspergillus antibodies in 93%. Eleven episodes were breakthrough infections. Antifungal treatment was prolonged, with a median of 13 months, and 6 patients (7 episodes) required surgery, with a high rate of postsurgical complications. One patient died and 6 had a relapse. Chronic and allergic forms of aspergillosis occurred in 17.5% of STAT3-deficient patients, mostly in lung cavities. Almost half had recurrences, despite prolonged antifungal treatment and/or surgery.

Sections du résumé

BACKGROUND
Autosomal-dominant signal transducer and activator of transcription 3 (STAT3) deficiency predisposes to recurrent bacterial pneumonia, complicated by bronchiectasis and cavitations. Aspergillosis is a major cause of morbidity in these patients. However, its diagnosis, classification, and treatment are challenging.
OBJECTIVE
We aimed to assess the prevalence and describe the clinical, mycological, and radiological presentation and related therapy and outcome of Aspergillus infections of the respiratory tract in the STAT3-deficient patients of the National French cohort.
METHODS
We performed a retrospective study of all pulmonary aspergillosis cases in STAT3-deficient patients (n = 74). Clinical and mycological data were collected up to October 2015 and imaging was centralized.
RESULTS
Twenty-one episodes of pulmonary aspergillosis in 13 (17.5%) STAT3-deficient patients were identified. The median age at first episode was 13 years (interquartile range, 10-26 years). Ninety percent of patients had previous bronchiectasis or cavitations. Infections were classified as follows: 5 single aspergilloma, 9 chronic cavity pulmonary aspergillosis, 5 allergic bronchopulmonary aspergillosis-like disease, and 2 mixed forms of concomitant allergic bronchopulmonary aspergillosis-like disease and chronic cavity pulmonary aspergillosis. No invasive aspergillosis cases were identified. Aspergillus species were isolated in 71% of episodes and anti-Aspergillus antibodies in 93%. Eleven episodes were breakthrough infections. Antifungal treatment was prolonged, with a median of 13 months, and 6 patients (7 episodes) required surgery, with a high rate of postsurgical complications. One patient died and 6 had a relapse.
CONCLUSIONS
Chronic and allergic forms of aspergillosis occurred in 17.5% of STAT3-deficient patients, mostly in lung cavities. Almost half had recurrences, despite prolonged antifungal treatment and/or surgery.

Identifiants

pubmed: 30878710
pii: S2213-2198(19)30263-6
doi: 10.1016/j.jaip.2019.02.041
pii:
doi:

Substances chimiques

Antifungal Agents 0
STAT3 Transcription Factor 0
STAT3 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1986-1995.e3

Investigateurs

H Coignard (H)
K Amazzough (K)
F Suarez (F)
S Blanche (S)
B Sendid (B)
M Cornu (M)
J F Bervar (JF)
A Deschildre (A)
L Wemeau (L)
C Fieschi (C)
A Alanio (A)
C Menetrey (C)
A Senechal (A)
F Ader (F)
P Tattevin (P)
C Pison (C)
L Grandiere-Perez (L)
D Garcia-Hermoso (D)
F Botterel-Chartier (F)

Informations de copyright

Copyright © 2019 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

Auteurs

Amélie Duréault (A)

Service de Maladies Infectieuses et Tropicales, Hôpital Necker-Enfants Malades, Assistance Publique-Hôpitaux de Paris (APHP), Université Paris Descartes, Paris, France.

Colas Tcherakian (C)

Service de Pneumologie, Hôpital Foch, Suresnes, France; Faculté des Sciences de la Santé Simone Veil, Université Versailles-Saint-Quentin-en-Yvelines, Versailles, France; National Referral Center for Hypereosinophilic (CEREO).

Sylvain Poiree (S)

Service de Radiologie, Hôpital Necker-Enfants Malades, APHP, Paris, France.

Emilie Catherinot (E)

Service de Pneumologie, Hôpital Foch, Suresnes, France; Faculté des Sciences de la Santé Simone Veil, Université Versailles-Saint-Quentin-en-Yvelines, Versailles, France.

François Danion (F)

Service de Maladies Infectieuses et Tropicales, Hôpital Necker-Enfants Malades, Assistance Publique-Hôpitaux de Paris (APHP), Université Paris Descartes, Paris, France.

Grégory Jouvion (G)

Unité de Neuropathologie Expérimentale, Institut Pasteur, Paris, France; Département de Génétique Médicale, Hôpital Trousseau, Sorbonne Université, APHP, Paris, France.

Marie Elisabeth Bougnoux (ME)

Service de Microbiologie, Hôpital Necker-Enfants Malades, APHP, Paris, France.

Nizar Mahlaoui (N)

Centre d'Etude des Déficits Immunitaires (CEDI), Hôpital Necker-Enfants Malades, APHP, Paris, France; CEREDIH, Centre de Référence des Déficits Immunitaires Héréditaires, Hôpital Universitaire Necker-Enfants Malades, APHP, Paris, France; Service Immunologie-Hématologie Pédiatrique, Hôpital Necker-Enfants Malades, APHP, Paris, France; Imagine Institut INSERM UMR1163, Université Paris Descartes, Paris, France.

Claire Givel (C)

Service de Pneumologie, Hôpital Foch, Suresnes, France; Faculté des Sciences de la Santé Simone Veil, Université Versailles-Saint-Quentin-en-Yvelines, Versailles, France.

Martin Castelle (M)

Service d'Hématologie, Hôpital Necker-Enfants Malades, APHP, Paris, France.

Capucine Picard (C)

Centre d'Etude des Déficits Immunitaires (CEDI), Hôpital Necker-Enfants Malades, APHP, Paris, France; CEREDIH, Centre de Référence des Déficits Immunitaires Héréditaires, Hôpital Universitaire Necker-Enfants Malades, APHP, Paris, France; Service Immunologie-Hématologie Pédiatrique, Hôpital Necker-Enfants Malades, APHP, Paris, France; Imagine Institut INSERM UMR1163, Université Paris Descartes, Paris, France.

Marie Olivia Chansdesris (MO)

Service d'Hématologie, Hôpital Necker-Enfants Malades, APHP, Paris, France.

Olivier Lortholary (O)

Service de Maladies Infectieuses et Tropicales, Hôpital Necker-Enfants Malades, Assistance Publique-Hôpitaux de Paris (APHP), Université Paris Descartes, Paris, France; Institut Pasteur, CNRS, Centre National de Référence Mycoses Invasives et Antifongiques, Unité de Mycologie Moléculaire, Paris, France.

Fanny Lanternier (F)

Service de Maladies Infectieuses et Tropicales, Hôpital Necker-Enfants Malades, Assistance Publique-Hôpitaux de Paris (APHP), Université Paris Descartes, Paris, France; Institut Pasteur, CNRS, Centre National de Référence Mycoses Invasives et Antifongiques, Unité de Mycologie Moléculaire, Paris, France. Electronic address: fanny.lanternier@aphp.fr.

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