Lesion Index Titration Using Contact-Force Technology Enables Safe and Effective Radiofrequency Lesion Creation at the Root of the Aorta and Pulmonary Artery.


Journal

Circulation. Arrhythmia and electrophysiology
ISSN: 1941-3084
Titre abrégé: Circ Arrhythm Electrophysiol
Pays: United States
ID NLM: 101474365

Informations de publication

Date de publication:
03 2019
Historique:
entrez: 19 3 2019
pubmed: 19 3 2019
medline: 20 12 2019
Statut: ppublish

Résumé

Ablation of some myocardial substrates requires catheter-based radiofrequency delivery at the root of a great artery. We studied the safety and efficacy parameters associated with catheter-based radiofrequency delivery at the root of the aorta and pulmonary artery. Thirty-six pigs underwent in-vivo catheter-based ablation under continuous contact-force and lesion index (power, contact-force, and time) monitoring during 60-s radiofrequency delivery with an open-irrigated tip catheter. Twenty-eight animals were allocated to groups receiving 40 W (n=9), 50 W (n=10), or 60 W (n=9) radiofrequency energy, and acute (n=22) and chronic (n=6) arterial wall damage was quantified by multiphoton microscopy in ex vivo samples. Adjacent myocardial lesions were quantified in parallel samples. The remaining 8 pigs were used to validate safety and efficacy parameters. Acute collagen and elastin alterations were significantly associated with radiofrequency power, although chronic assessment revealed vascular wall recovery in lesions without steam pop. The main parameters associated with steam pops were median peak temperature >42°C and impedance falls >23 ohms. Unlike other parameters, lesion index values of 9.1 units (interquartile range, 8.7-9.8) were associated with the presence of adjacent myocardial lesions in both univariate ( P=0.03) and multivariate analyses ( P=0.049; odds ratio, 1.99; 95% CI, 1.02-3.98). In the validation group, lesion index values using 40 W over a range of contact-forces correlated with the size of radiofrequency lesions (R Lesion index values obtained during 40 W radiofrequency applications reliably monitor safe and effective lesion creation at the root of the great arteries.

Sections du résumé

BACKGROUND
Ablation of some myocardial substrates requires catheter-based radiofrequency delivery at the root of a great artery. We studied the safety and efficacy parameters associated with catheter-based radiofrequency delivery at the root of the aorta and pulmonary artery.
METHODS
Thirty-six pigs underwent in-vivo catheter-based ablation under continuous contact-force and lesion index (power, contact-force, and time) monitoring during 60-s radiofrequency delivery with an open-irrigated tip catheter. Twenty-eight animals were allocated to groups receiving 40 W (n=9), 50 W (n=10), or 60 W (n=9) radiofrequency energy, and acute (n=22) and chronic (n=6) arterial wall damage was quantified by multiphoton microscopy in ex vivo samples. Adjacent myocardial lesions were quantified in parallel samples. The remaining 8 pigs were used to validate safety and efficacy parameters.
RESULTS
Acute collagen and elastin alterations were significantly associated with radiofrequency power, although chronic assessment revealed vascular wall recovery in lesions without steam pop. The main parameters associated with steam pops were median peak temperature >42°C and impedance falls >23 ohms. Unlike other parameters, lesion index values of 9.1 units (interquartile range, 8.7-9.8) were associated with the presence of adjacent myocardial lesions in both univariate ( P=0.03) and multivariate analyses ( P=0.049; odds ratio, 1.99; 95% CI, 1.02-3.98). In the validation group, lesion index values using 40 W over a range of contact-forces correlated with the size of radiofrequency lesions (R
CONCLUSIONS
Lesion index values obtained during 40 W radiofrequency applications reliably monitor safe and effective lesion creation at the root of the great arteries.

Identifiants

pubmed: 30879334
doi: 10.1161/CIRCEP.118.007080
pmc: PMC6426438
mid: NIHMS1521758
doi:

Substances chimiques

Collagen 9007-34-5
Elastin 9007-58-3

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e007080

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL060843
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL080159
Pays : United States

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Auteurs

José Manuel Alfonso-Almazán (JM)

Centro Nacional de Investigaciones Cardiovasculares, Carlos III (CNIC), Myocardial Pathophysiology Area (J.M.A.-A., J.G.Q., S.L.-C., J.J., D.F.-R.).

Jorge G Quintanilla (JG)

Centro Nacional de Investigaciones Cardiovasculares, Carlos III (CNIC), Myocardial Pathophysiology Area (J.M.A.-A., J.G.Q., S.L.-C., J.J., D.F.-R.).
Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).

María Jesús García-Torrent (MJ)

Fundación Interhospitalaria para la Investigación Cardiovascular (FIC) (M.J.G.-T., J.P.-V.).

Santiago Laguna-Castro (S)

Centro Nacional de Investigaciones Cardiovasculares, Carlos III (CNIC), Myocardial Pathophysiology Area (J.M.A.-A., J.G.Q., S.L.-C., J.J., D.F.-R.).

Cruz Rodríguez-Bobada (C)

Experimental Medicine and Surgery Unit, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (C.R.-B., P.G.).

Pablo González (P)

Experimental Medicine and Surgery Unit, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (C.R.-B., P.G.).

Juan José González-Ferrer (JJ)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).

Pablo Salinas (P)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).

Victoria Cañadas-Godoy (V)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).

Javier Moreno (J)

CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).
Hospital Universitario Ramón y Cajal, Department of Cardiology, Madrid, Spain (J.M.).

Luis Borrego-Bernabé (L)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).

Nicasio Pérez-Castellano (N)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).

José Jalife (J)

Centro Nacional de Investigaciones Cardiovasculares, Carlos III (CNIC), Myocardial Pathophysiology Area (J.M.A.-A., J.G.Q., S.L.-C., J.J., D.F.-R.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).
Center for Arrhythmia Research, Cardiovascular Research Center, Department of Internal Medicine, University of Michigan, Ann Arbor (J.J.).

Julián Perez-Villacastín (J)

Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).
Fundación Interhospitalaria para la Investigación Cardiovascular (FIC) (M.J.G.-T., J.P.-V.).

David Filgueiras-Rama (D)

Centro Nacional de Investigaciones Cardiovasculares, Carlos III (CNIC), Myocardial Pathophysiology Area (J.M.A.-A., J.G.Q., S.L.-C., J.J., D.F.-R.).
Cardiovascular Institute, Department of Cardiology, Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC) (J.G.Q., J.J.G.-F., P.S., V.C.-G., L.B.-B., N.P.-C., J.P.-V.).
CIBER de Enfermedades Cardiovasculares (J.G.Q., J.J.G.-F., V.C.-G., J.M., N.P.-C., J.J., J.P.-V., D.F.-R.).

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