Elevated doublecortin-like kinase 1 serum levels revert to baseline after therapy in early stage esophageal adenocarcinoma.

Cancer stem cell DCLK1 Doublecortin-like kinase 1 ELISA Esophageal adenocarcinoma

Journal

Biomarker research
ISSN: 2050-7771
Titre abrégé: Biomark Res
Pays: England
ID NLM: 101607860

Informations de publication

Date de publication:
2019
Historique:
received: 12 12 2018
accepted: 27 02 2019
entrez: 23 3 2019
pubmed: 23 3 2019
medline: 23 3 2019
Statut: epublish

Résumé

Barrett's esophagus (BE) and esophageal adenocarcinoma (EAC) incidence has been increasing in the United States for greater than 30 years. For the majority of EAC patients, treatment is limited and prognosis poor. Doublecortin like kinase-1 (DCLK1) is a cancer stem cell marker with elevated expression in BE patients with high grade dysplasia and/or EAC. This prospective cohort study was designed to compare serum DCLK1 levels before and after EAC treatment with endoscopic mucosal resection (EMR) and/or radio-frequency ablation (RFA). Barrett's esophagus patients with low or high-grade dysplasia ( Serum DCLK1 levels were increased by > 50% in Barrett's Esophagus ( EAC treatment results in significantly decreased serum DCLK1 levels, suggesting that DCLK1 may be useful as a non-invasive disease regression biomarker following treatment. Biomarkers for EAC therapeutic response have been poorly studied and no reliable marker has been discovered thus far. These results demonstrate that DCLK1 may have potential as a circulating biomarker of the response to therapy in EAC, which could be used to improve patient outcomes.

Sections du résumé

BACKGROUND BACKGROUND
Barrett's esophagus (BE) and esophageal adenocarcinoma (EAC) incidence has been increasing in the United States for greater than 30 years. For the majority of EAC patients, treatment is limited and prognosis poor. Doublecortin like kinase-1 (DCLK1) is a cancer stem cell marker with elevated expression in BE patients with high grade dysplasia and/or EAC. This prospective cohort study was designed to compare serum DCLK1 levels before and after EAC treatment with endoscopic mucosal resection (EMR) and/or radio-frequency ablation (RFA).
METHODS METHODS
Barrett's esophagus patients with low or high-grade dysplasia (
RESULTS RESULTS
Serum DCLK1 levels were increased by > 50% in Barrett's Esophagus (
CONCLUSIONS CONCLUSIONS
EAC treatment results in significantly decreased serum DCLK1 levels, suggesting that DCLK1 may be useful as a non-invasive disease regression biomarker following treatment.
IMPACT CONCLUSIONS
Biomarkers for EAC therapeutic response have been poorly studied and no reliable marker has been discovered thus far. These results demonstrate that DCLK1 may have potential as a circulating biomarker of the response to therapy in EAC, which could be used to improve patient outcomes.

Identifiants

pubmed: 30899515
doi: 10.1186/s40364-019-0157-z
pii: 157
pmc: PMC6408757
doi:

Types de publication

Journal Article

Langues

eng

Pagination

5

Déclaration de conflit d'intérêts

All patients provided informed consent and samples were deidentified. The University of Oklahoma Health Sciences Center Institutional Review Board approved the study.Not applicable.NW and CWH have ownership interests in COARE Holdings Inc.Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

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Auteurs

Emily M Christman (EM)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.

Parthasarathy Chandrakesan (P)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.
2Peggy and Charles Stephenson Cancer Center, Oklahoma City, OK USA.

Nathaniel Weygant (N)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.

John T Maple (JT)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.

William M Tierney (WM)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.

Kenneth J Vega (KJ)

3Department of Medicine, Division of Gastroenterology and Hepatology, Augusta University-Medical College of Georgia, 1120 15th Street, AD 2226, Augusta, GA 30912 USA.

Courtney W Houchen (CW)

1Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK USA.
2Peggy and Charles Stephenson Cancer Center, Oklahoma City, OK USA.

Classifications MeSH