Hydrogen sulfide-mediated regulation of cell death signaling ameliorates adverse cardiac remodeling and diabetic cardiomyopathy.
apoptosis
autophagy
diabetes
necroptosis
pyroptosis
therapeutic strategy
Journal
American journal of physiology. Heart and circulatory physiology
ISSN: 1522-1539
Titre abrégé: Am J Physiol Heart Circ Physiol
Pays: United States
ID NLM: 100901228
Informations de publication
Date de publication:
01 06 2019
01 06 2019
Historique:
pubmed:
30
3
2019
medline:
17
3
2020
entrez:
30
3
2019
Statut:
ppublish
Résumé
The death of cardiomyocytes is a precursor for the cascade of hypertrophic and fibrotic remodeling that leads to cardiomyopathy. In diabetes mellitus (DM), the metabolic environment of hyperglycemia, hyperlipidemia, and oxidative stress causes cardiomyocyte cell death, leading to diabetic cardiomyopathy (DMCM), an independent cause of heart failure. Understanding the roles of the cell death signaling pathways involved in the development of cardiomyopathies is crucial to the discovery of novel targeted therapeutics and biomarkers for DMCM. Recent evidence suggests that hydrogen sulfide (H
Identifiants
pubmed: 30925069
doi: 10.1152/ajpheart.00004.2019
pmc: PMC6620689
doi:
Substances chimiques
Cardiovascular Agents
0
Hydrogen Sulfide
YY9FVM7NSN
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
H1237-H1252Subventions
Organisme : NHLBI NIH HHS
ID : R01 HL113281
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL116205
Pays : United States
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