Identification of 19 Novel Hepatitis C Virus Subtypes-Further Expanding HCV Classification.

direct-acting antivirals (DAAs) phylogenetic analysis resistance-associated substitutions (RAS) sofosbuvir velpatasvir voxilaprevir

Journal

Open forum infectious diseases
ISSN: 2328-8957
Titre abrégé: Open Forum Infect Dis
Pays: United States
ID NLM: 101637045

Informations de publication

Date de publication:
Mar 2019
Historique:
received: 03 01 2019
revised: 01 02 2019
accepted: 19 02 2019
entrez: 6 4 2019
pubmed: 6 4 2019
medline: 6 4 2019
Statut: epublish

Résumé

Hepatitis C virus (HCV) is currently classified into 8 genotypes and 86 subtypes. The objective of this study was to characterize novel HCV subtypes and to investigate the impact of subtypes on treatment outcome. Full-genome sequencing was performed on HCV plasma samples with <85% sequence homology of NS3, NS5A, and/or NS5B to HCV genotype (GT) 1-8 reference strains. A total of 14 653 patients with GT1-6 HCV infection were enrolled in clinical studies of sofosbuvir-based regimens. For the majority of the patients, a specific subtype could be assigned based on a close genetic relationship to previously described subtypes. However, for 19 patients, novel subtypes were identified with <85% homology compared with previously described subtypes. These novel subtypes had the following genotypes: 9 in GT2, 5 in GT4, 2 in GT6, and 1 each in GT1, GT3, and GT5. Despite the presence of polymorphisms at resistance-associated substitution positions, 18 of the 19 patients treated with sofosbuvir-containing therapy achieved SVR12. Nineteen novel HCV subtypes were identified, suggesting an even greater genetic diversity of HCV subtypes than previously recognized.

Sections du résumé

BACKGROUND BACKGROUND
Hepatitis C virus (HCV) is currently classified into 8 genotypes and 86 subtypes. The objective of this study was to characterize novel HCV subtypes and to investigate the impact of subtypes on treatment outcome.
METHODS METHODS
Full-genome sequencing was performed on HCV plasma samples with <85% sequence homology of NS3, NS5A, and/or NS5B to HCV genotype (GT) 1-8 reference strains.
RESULTS RESULTS
A total of 14 653 patients with GT1-6 HCV infection were enrolled in clinical studies of sofosbuvir-based regimens. For the majority of the patients, a specific subtype could be assigned based on a close genetic relationship to previously described subtypes. However, for 19 patients, novel subtypes were identified with <85% homology compared with previously described subtypes. These novel subtypes had the following genotypes: 9 in GT2, 5 in GT4, 2 in GT6, and 1 each in GT1, GT3, and GT5. Despite the presence of polymorphisms at resistance-associated substitution positions, 18 of the 19 patients treated with sofosbuvir-containing therapy achieved SVR12.
CONCLUSIONS CONCLUSIONS
Nineteen novel HCV subtypes were identified, suggesting an even greater genetic diversity of HCV subtypes than previously recognized.

Identifiants

pubmed: 30949527
doi: 10.1093/ofid/ofz076
pii: ofz076
pmc: PMC6440686
doi:

Types de publication

Journal Article

Langues

eng

Pagination

ofz076

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Auteurs

Charlotte Hedskog (C)

Gilead Sciences Inc, Foster City, California.

Bandita Parhy (B)

Gilead Sciences Inc, Foster City, California.

Silvia Chang (S)

Gilead Sciences Inc, Foster City, California.

Stefan Zeuzem (S)

Johann Wolfgang Goethe University Hospital, Frankfurt, Germany.

Christophe Moreno (C)

CUB Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium.

Stephen D Shafran (SD)

University of Alberta, Edmonton, Canada.

Sergio M Borgia (SM)

William Osler Health System, Brampton, Ontario, Canada.

Tarik Asselah (T)

INSERM UMR 1149, AP-HP Hôpital Beaujon, Université Paris Diderot and Centre de Recherche sur l'Inflammation, Clichy, France.

Laurent Alric (L)

Service de Médecine Interne, Toulouse Cedex, France.

Armand Abergel (A)

CHU Estaing, Clermont Ferrand, France.

Jyh-Jou Chen (JJ)

Chi Mei Hospital, Tainan, Taiwan.

Jane Collier (J)

John Radcliffe Hospital, Oxford, UK.

Dharmesh Kapoor (D)

Global Hospitals Group, Hyderabad, India.

Robert H Hyland (RH)

Gilead Sciences Inc, Foster City, California.

Peter Simmonds (P)

Nuffield Department of Medicine, University of Oxford, Oxford, UK.

Hongmei Mo (H)

Gilead Sciences Inc, Foster City, California.

Evguenia S Svarovskaia (ES)

Gilead Sciences Inc, Foster City, California.

Classifications MeSH