MRI Assessment of Oxygen Metabolism and Hemodynamic Status in Symptomatic Intracranial Atherosclerotic Stenosis: A Pilot Study.


Journal

Journal of neuroimaging : official journal of the American Society of Neuroimaging
ISSN: 1552-6569
Titre abrégé: J Neuroimaging
Pays: United States
ID NLM: 9102705

Informations de publication

Date de publication:
07 2019
Historique:
received: 19 11 2018
revised: 14 03 2019
accepted: 17 03 2019
pubmed: 6 4 2019
medline: 17 6 2020
entrez: 6 4 2019
Statut: ppublish

Résumé

Hemodynamic and metabolic impairment in intracranial atherosclerotic stenosis (ICAS) may promote stroke vulnerability particularly in borderzone areas. Perfusion and oxygen mapping magnetic resonance imaging (MRI) may provide useful information in this setting. In this pilot study, patients with symptomatic atherosclerotic anterior circulation stenosis ≥60%, without other sources of ischemic stroke, were included. High-resolution vessel wall MRI quantified the stenosis degree, and hemodynamic and metabolic impairment was assessed at baseline using dynamic susceptibility contrast perfusion and multiparametric quantitative blood-oxygen-level-dependent (mqBOLD) oxygenation MRI. All parameters were assessed within both hemispheres and in borderzone areas. Forty-three subjects with intracranial artery narrowing were screened from November 2014 to January 2016. Eleven patients met the study criteria (mean ± standard deviation age = 64.4 ± 10.6 years, the mean degree of stenosis was 76.9 ± 23.4%). No interhemispheric differences were observed across oxygen (cerebral metabolic rate of oxygen and tissular saturation of oxygen) or perfusion (mean transit time, time to maximum, T Symptomatic ICAS had no global impact on perfusion and oxygen mapping MRI at resting state. A significant increase in nCBV was found within anterior borderzone areas.

Sections du résumé

BACKGROUND AND PURPOSE
Hemodynamic and metabolic impairment in intracranial atherosclerotic stenosis (ICAS) may promote stroke vulnerability particularly in borderzone areas. Perfusion and oxygen mapping magnetic resonance imaging (MRI) may provide useful information in this setting.
METHODS
In this pilot study, patients with symptomatic atherosclerotic anterior circulation stenosis ≥60%, without other sources of ischemic stroke, were included. High-resolution vessel wall MRI quantified the stenosis degree, and hemodynamic and metabolic impairment was assessed at baseline using dynamic susceptibility contrast perfusion and multiparametric quantitative blood-oxygen-level-dependent (mqBOLD) oxygenation MRI. All parameters were assessed within both hemispheres and in borderzone areas.
RESULTS
Forty-three subjects with intracranial artery narrowing were screened from November 2014 to January 2016. Eleven patients met the study criteria (mean ± standard deviation age = 64.4 ± 10.6 years, the mean degree of stenosis was 76.9 ± 23.4%). No interhemispheric differences were observed across oxygen (cerebral metabolic rate of oxygen and tissular saturation of oxygen) or perfusion (mean transit time, time to maximum, T
CONCLUSION
Symptomatic ICAS had no global impact on perfusion and oxygen mapping MRI at resting state. A significant increase in nCBV was found within anterior borderzone areas.

Identifiants

pubmed: 30950158
doi: 10.1111/jon.12615
doi:

Substances chimiques

Oxygen S88TT14065

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

467-475

Informations de copyright

© 2019 by the American Society of Neuroimaging.

Auteurs

Omer F Eker (OF)

Department of Neuroradiology, Hospices Civils de Lyon, Bron, France.
CREATIS CNRS UMR 5220, INSERM U1044, Villeurbanne cedex, France.

Roxana Ameli (R)

Department of Neuroradiology, Hospices Civils de Lyon, Bron, France.

Nikolaos Makris (N)

CREATIS CNRS UMR 5220, INSERM U1044, Villeurbanne cedex, France.

Thomas Jurkovic (T)

Department of Neuroradiology, Hospices Civils de Lyon, Bron, France.

Olivier Montigon (O)

INSERM U1216, Grenoble Institut des Neurosciences, La Tronche, France.

Emmanuel L Barbier (EL)

INSERM U1216, Grenoble Institut des Neurosciences, La Tronche, France.
Université Grenoble Alpes, Saint-Martin-d'Hères, France.

Tae Hee Cho (TH)

CREATIS CNRS UMR 5220, INSERM U1044, Villeurbanne cedex, France.

Norbert Nighoghossian (N)

CREATIS CNRS UMR 5220, INSERM U1044, Villeurbanne cedex, France.

Yves Berthezène (Y)

Department of Neuroradiology, Hospices Civils de Lyon, Bron, France.
CREATIS CNRS UMR 5220, INSERM U1044, Villeurbanne cedex, France.
Department of Vascular Neurology, Hospices Civils de Lyon, Hôpital Pierre Wertheimer, Bron, France.

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