Modulation of mitochondrial dysfunction for treatment of disease.


Journal

Bioorganic & medicinal chemistry letters
ISSN: 1464-3405
Titre abrégé: Bioorg Med Chem Lett
Pays: England
ID NLM: 9107377

Informations de publication

Date de publication:
01 06 2019
Historique:
received: 07 01 2019
revised: 21 03 2019
accepted: 25 03 2019
pubmed: 8 4 2019
medline: 12 9 2020
entrez: 8 4 2019
Statut: ppublish

Résumé

Mitochondrial dysfunction is a causative and/or exacerbating feature of many pathologies. We discuss below approaches to modulate mitochondrial dysfunction that involve (1) increasing their energetic efficiency by targeting gene expression regulators such as PPAR or AMPK, (2) using antioxidant compounds to reduce the toxic reactive oxygen species mitochondria produce under stress, or (3) modulating aspects on the innate mitochondrial quality control system. The latter comprise linked processes of biogenesis, dynamic morphological changes, and elimination of defective mitochondria by mitophagy. We discuss representative compounds in all three classes.

Identifiants

pubmed: 30954429
pii: S0960-894X(19)30180-5
doi: 10.1016/j.bmcl.2019.03.041
pii:
doi:

Substances chimiques

Antioxidants 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1270-1277

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier Ltd.. All rights reserved.

Auteurs

Michael Webb (M)

Mitobridge Inc, an Astellas Company, 1030 Massachusetts Ave, Cambridge, MA 02138, USA. Electronic address: michael.webb@astellas.com.

Dionisia P Sideris (DP)

Mitobridge Inc, an Astellas Company, 1030 Massachusetts Ave, Cambridge, MA 02138, USA. Electronic address: jeni.sideris@astellas.com.

Margaret Biddle (M)

Mitobridge Inc, an Astellas Company, 1030 Massachusetts Ave, Cambridge, MA 02138, USA. Electronic address: margaret.Biddle@astellas.com.

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Classifications MeSH