Atrophic and hypertrophic photoaging: Clinical, histologic, and molecular features of 2 distinct phenotypes of photoaged skin.


Journal

Journal of the American Academy of Dermatology
ISSN: 1097-6787
Titre abrégé: J Am Acad Dermatol
Pays: United States
ID NLM: 7907132

Informations de publication

Date de publication:
Aug 2019
Historique:
received: 03 08 2018
revised: 17 01 2019
accepted: 08 03 2019
pubmed: 8 4 2019
medline: 21 12 2019
entrez: 8 4 2019
Statut: ppublish

Résumé

Exposure to the sun causes premature skin aging, known as photoaging. Clinical features of photoaging vary widely among individuals. In one form, skin appears thin with telangiectasia, and in another form, skin appears thickened with coarse wrinkles. Etiologic, clinical, and therapeutic distinctions among different forms of photoaging remain largely unknown. To characterize the clinical, histologic, and molecular features of hypertrophic and atrophic photoaging. In total, 53 individuals were clinically classified as having primarily atrophic or hypertrophic photoaging or neither (controls). Participants' demographic and sun exposure-related lifestyle data were captured by questionnaire. Fifteen clinical features of participants were qualitatively or quantitively scored. Facial biopsies were analyzed for gene expression and histologic characteristics. Actinic and seborrheic keratosis, telangiectasia, and prior incidence of skin cancers were statistically significantly greater and photoaging scale severity, coarse wrinkles, thickness, and sallowness were significantly reduced in atrophic versus hypertrophic groups. Histology also revealed significantly less elastotic material in atrophic photoaging. Gene expression of matrix metalloproteinases and collagens did not differ between the 2 forms of photoaging. The study was not designed to identify other possible subtypes of photoaging. Systematic, categorical, and quantitative clinical and histologic assessments distinguish atrophic and hypertrophic photoaging.

Sections du résumé

BACKGROUND BACKGROUND
Exposure to the sun causes premature skin aging, known as photoaging. Clinical features of photoaging vary widely among individuals. In one form, skin appears thin with telangiectasia, and in another form, skin appears thickened with coarse wrinkles. Etiologic, clinical, and therapeutic distinctions among different forms of photoaging remain largely unknown.
OBJECTIVE OBJECTIVE
To characterize the clinical, histologic, and molecular features of hypertrophic and atrophic photoaging.
METHODS METHODS
In total, 53 individuals were clinically classified as having primarily atrophic or hypertrophic photoaging or neither (controls). Participants' demographic and sun exposure-related lifestyle data were captured by questionnaire. Fifteen clinical features of participants were qualitatively or quantitively scored. Facial biopsies were analyzed for gene expression and histologic characteristics.
RESULTS RESULTS
Actinic and seborrheic keratosis, telangiectasia, and prior incidence of skin cancers were statistically significantly greater and photoaging scale severity, coarse wrinkles, thickness, and sallowness were significantly reduced in atrophic versus hypertrophic groups. Histology also revealed significantly less elastotic material in atrophic photoaging. Gene expression of matrix metalloproteinases and collagens did not differ between the 2 forms of photoaging.
LIMITATIONS CONCLUSIONS
The study was not designed to identify other possible subtypes of photoaging.
CONCLUSION CONCLUSIONS
Systematic, categorical, and quantitative clinical and histologic assessments distinguish atrophic and hypertrophic photoaging.

Identifiants

pubmed: 30954583
pii: S0190-9622(19)30528-6
doi: 10.1016/j.jaad.2019.03.081
pii:
doi:

Substances chimiques

Collagen 9007-34-5
Matrix Metalloproteinases EC 3.4.24.-

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

480-488

Informations de copyright

Copyright © 2019. Published by Elsevier Inc.

Auteurs

Dana L Sachs (DL)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan. Electronic address: dsachs@med.umich.edu.

James Varani (J)

Department of Pathology, University of Michigan, Ann Arbor, Michigan.

Heather Chubb (H)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

Suzanne E G Fligiel (SEG)

Department of Pathology, University of Michigan, Ann Arbor, Michigan.

Yilei Cui (Y)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

Ken Calderone (K)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

Yolanda Helfrich (Y)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

Gary J Fisher (GJ)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

John J Voorhees (JJ)

Department of Dermatology, University of Michigan, Ann Arbor, Michigan.

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Classifications MeSH