Difficulties with emotion regulation as a transdiagnostic mechanism linking child maltreatment with the emergence of psychopathology.
adversity
attention bias
p factor
rumination
threat
Journal
Development and psychopathology
ISSN: 1469-2198
Titre abrégé: Dev Psychopathol
Pays: United States
ID NLM: 8910645
Informations de publication
Date de publication:
08 2019
08 2019
Historique:
pubmed:
9
4
2019
medline:
15
11
2019
entrez:
9
4
2019
Statut:
ppublish
Résumé
Childhood maltreatment is associated with increased risk for most forms of psychopathology. We examine emotion dysregulation as a transdiagnostic mechanism linking maltreatment with general psychopathology. A sample of 262 children and adolescents participated; 162 (61.8%) experienced abuse or exposure to domestic violence. We assessed four emotion regulation processes (cognitive reappraisal, attention bias to threat, expressive suppression, and rumination) and emotional reactivity. Psychopathology symptoms were assessed concurrently and at a 2-year longitudinal follow-up. A general psychopathology factor (p factor), representing co-occurrence of psychopathology symptoms across multiple internalizing and externalizing domains, was estimated using confirmatory factor analysis. Maltreatment was associated with heightened emotional reactivity and greater use of expressive suppression and rumination. The association of maltreatment with attention bias varied across development, with maltreated children exhibiting a bias toward threat and adolescents a bias away from threat. Greater emotional reactivity and engagement in rumination mediated the longitudinal association between maltreatment and increased general psychopathology over time. Emotion dysregulation following childhood maltreatment occurs at multiple stages of the emotion generation process, in some cases varies across development, and serves as a transdiagnostic mechanism linking child maltreatment with general psychopathology.
Identifiants
pubmed: 30957738
pii: S0954579419000348
doi: 10.1017/S0954579419000348
pmc: PMC6620140
mid: NIHMS1022685
doi:
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
899-915Subventions
Organisme : NIMH NIH HHS
ID : K01 MH116325
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH103291
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH106482
Pays : United States
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