D1-Dopamine Receptor Availability in First-Episode Neuroleptic Naive Psychosis Patients.


Journal

The international journal of neuropsychopharmacology
ISSN: 1469-5111
Titre abrégé: Int J Neuropsychopharmacol
Pays: England
ID NLM: 9815893

Informations de publication

Date de publication:
01 07 2019
Historique:
received: 10 01 2019
revised: 12 03 2019
accepted: 05 04 2019
pubmed: 9 4 2019
medline: 9 4 2020
entrez: 9 4 2019
Statut: ppublish

Résumé

Positron emission tomography studies examining differences in D1-dopamine receptor binding between control subjects and patients with schizophrenia have been inconsistent, reporting higher, lower, and no difference in the frontal cortex. Exposure to antipsychotic medication has been suggested to be a likely source of this heterogeneity, and thus there is a need for studies of patients at early stages of the disorder who have not been exposed to such drugs. Here, we compared 17 healthy control subjects and 18 first-episode neuroleptic naive patients with schizophrenia or schizophreniform psychosis using positron emission tomography and the D1-dopamine receptor radioligand [11C]SCH23390. We observed a statistically significant difference in the dorsolateral prefrontal cortex. Contrary to our expectations, patients had less D1-dopamine receptor availability with a moderate effect size. In a Bayesian analysis, we show that the data are over 50 times more likely to have occurred under the decrease as opposed to the increase hypothesis. This effect was not global, as our analysis showed that the null hypothesis was preferred over either hypothesis in the striatum. This investigation represents the largest single sample of neuroleptic-naive patients examined for D1-dopamine receptor availability using PET and suggests a reduction of prefrontal D1-dopamine receptor density in the pathophysiology of schizophrenia. However, further work will be required to reach a consensus.

Sections du résumé

BACKGROUND
Positron emission tomography studies examining differences in D1-dopamine receptor binding between control subjects and patients with schizophrenia have been inconsistent, reporting higher, lower, and no difference in the frontal cortex. Exposure to antipsychotic medication has been suggested to be a likely source of this heterogeneity, and thus there is a need for studies of patients at early stages of the disorder who have not been exposed to such drugs.
METHODS
Here, we compared 17 healthy control subjects and 18 first-episode neuroleptic naive patients with schizophrenia or schizophreniform psychosis using positron emission tomography and the D1-dopamine receptor radioligand [11C]SCH23390.
RESULTS
We observed a statistically significant difference in the dorsolateral prefrontal cortex. Contrary to our expectations, patients had less D1-dopamine receptor availability with a moderate effect size. In a Bayesian analysis, we show that the data are over 50 times more likely to have occurred under the decrease as opposed to the increase hypothesis. This effect was not global, as our analysis showed that the null hypothesis was preferred over either hypothesis in the striatum.
CONCLUSIONS
This investigation represents the largest single sample of neuroleptic-naive patients examined for D1-dopamine receptor availability using PET and suggests a reduction of prefrontal D1-dopamine receptor density in the pathophysiology of schizophrenia. However, further work will be required to reach a consensus.

Identifiants

pubmed: 30958880
pii: 5432304
doi: 10.1093/ijnp/pyz017
pmc: PMC6600463
doi:

Substances chimiques

Benzazepines 0
DRD1 protein, human 0
Radiopharmaceuticals 0
Receptors, Dopamine D1 0
SCH 23390 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

415-425

Informations de copyright

© The Author(s) 2019. Published by Oxford University Press on behalf of CINP.

Références

Am J Psychiatry. 2006 Oct;163(10):1747-53
pubmed: 17012685
Acta Psychiatr Scand Suppl. 1986;326:1-37
pubmed: 3458353
J Nucl Med. 1998 May;39(5):792-7
pubmed: 9591577
Psychopharmacology (Berl). 1995 Oct;121(3):317-22
pubmed: 8584612
Science. 1991 Feb 22;251(4996):947-50
pubmed: 1825731
J Cereb Blood Flow Metab. 2001 Sep;21(9):1034-57
pubmed: 11524609
Arch Gen Psychiatry. 2012 Aug;69(8):776-86
pubmed: 22474070
Psychopharmacology (Berl). 1995 Oct;121(3):323-7
pubmed: 8584613
Psychopharmacology (Berl). 1991;103(1):41-5
pubmed: 1826059
Br J Psychiatry Suppl. 1992 May;(17):30-3
pubmed: 1358126
J Psychopharmacol. 2012 Jun;26(6):794-805
pubmed: 21768159
Neuroscience. 2010 Apr 28;167(1):104-10
pubmed: 20109534
Neurosci Lett. 2019 Jan 19;691:26-34
pubmed: 29518542
Schizophr Bull. 1988;14(2):157-68
pubmed: 3059465
Schizophr Bull. 2019 Apr 25;45(3):670-679
pubmed: 29878197
Biol Psychiatry. 1999 Jan 1;45(1):17-25
pubmed: 9894571
Soc Psychol Personal Sci. 2017 May;8(4):355-362
pubmed: 28736600
Cereb Cortex. 2000 Nov;10(11):1078-92
pubmed: 11053229
Hum Brain Mapp. 1994;1(3):173-84
pubmed: 24578038
Life Sci. 2000;66(25):2455-64
pubmed: 10894088
J Neurosci. 2002 May 1;22(9):3708-19
pubmed: 11978847
Life Sci. 2010 May 22;86(21-22):814-8
pubmed: 20361984
Perspect Psychol Sci. 2014 Nov;9(6):641-51
pubmed: 26186114
Mol Psychiatry. 2002;7(3):233, 322-8
pubmed: 11920160
Biochemistry. 1995 May 2;34(17):5960-70
pubmed: 7727453
EJNMMI Res. 2018 Aug 2;8(1):74
pubmed: 30069645
Psychiatry Res. 1995 Aug 8;61(2):67-83
pubmed: 7480390
Int J Rad Appl Instrum A. 1986;37(10):1039-43
pubmed: 3027000
J Psychopharmacol. 2016 May;30(5):428-35
pubmed: 26966119
Neurobiol Aging. 2011 Oct;32(10):1849-56
pubmed: 19962789
Neuropsychopharmacology. 2008 Sep;33(10):2435-41
pubmed: 18288096
Nature. 1997 Feb 13;385(6617):634-6
pubmed: 9024661
Proc Natl Acad Sci U S A. 1994 May 10;91(10):4353-6
pubmed: 8183912
Am J Psychiatry. 1999 Jan;156(1):72-8
pubmed: 9892300
Neuropsychopharmacology. 1994 Dec;11(4):245-56
pubmed: 7531978
Am J Psychiatry. 2000 Jun;157(6):1016-8
pubmed: 10831488
Biol Psychiatry. 1996 Dec 15;40(12):1191-9
pubmed: 8959283
Neuropsychopharmacology. 2003 Sep;28(9):1703-11
pubmed: 12813475
Science. 2000 Mar 17;287(5460):2020-2
pubmed: 10720329
Elife. 2017 Sep 05;6:
pubmed: 28870286
Arch Gen Psychiatry. 1992 Jul;49(7):538-44
pubmed: 1352677
Am J Psychiatry. 2002 May;159(5):761-7
pubmed: 11986129
J Neurophysiol. 1994 Feb;71(2):515-28
pubmed: 7909839
Psychopharmacology (Berl). 2013 Jul;228(1):167-74
pubmed: 23460265
Transl Psychiatry. 2016 Apr 12;6:e780
pubmed: 27070408
Eur Neuropsychopharmacol. 2016 Jul;26(7):1248-50
pubmed: 27179966
Psychopharmacology (Berl). 1992;107(1):23-9
pubmed: 1534178
Psychopharmacology (Berl). 1999 Sep;146(2):220-7
pubmed: 10525759
JAMA Psychiatry. 2015 Apr;72(4):316-24
pubmed: 25651194
Arch Gen Psychiatry. 2010 Jan;67(1):9-16
pubmed: 20048218
J Pharmacokinet Biopharm. 1987 Dec;15(6):657-80
pubmed: 3450848
Synapse. 1999 May;32(2):93-109
pubmed: 10231129
J Comput Assist Tomogr. 1994 Jan-Feb;18(1):110-8
pubmed: 8282858
J Pharmacol Exp Ther. 1997 Apr;281(1):597-603
pubmed: 9103549
Biol Psychiatry. 2010 Mar 15;67(6):575-80
pubmed: 20138255
Neuroimage. 2017 Jul 15;155:344-353
pubmed: 28419852
Mol Imaging Biol. 2007 May-Jun;9(3):117-25
pubmed: 17473957
J Comput Assist Tomogr. 1981 Feb;5(1):136-41
pubmed: 6972391
Lancet. 1995 Sep 16;346(8977):743-9
pubmed: 7658878
J Nucl Med. 1998 Dec;39(12):2061-8
pubmed: 9867142
Schizophr Res. 1998 May 4;31(1):13-7
pubmed: 9633832
Arch Gen Psychiatry. 2009 Aug;66(8):811-22
pubmed: 19652121
Neuroimage. 1996 Dec;4(3 Pt 1):153-8
pubmed: 9345505
Acta Psychiatr Scand. 1974;50(4):425-62
pubmed: 4423855
Neuroimage. 2005 Jul 1;26(3):839-51
pubmed: 15955494
Schizophr Bull. 2009 May;35(3):549-62
pubmed: 19325164
Hum Psychopharmacol. 2018 Sep;33(5):e2675
pubmed: 30306671
J Cereb Blood Flow Metab. 2007 Oct;27(10):1733-41
pubmed: 17311076
Biol Psychiatry. 2017 Jan 1;81(1):67-77
pubmed: 26946382
Science. 1979 Aug 31;205(4409):929-32
pubmed: 112679
Synapse. 1998 Sep;30(1):56-61
pubmed: 9704881
Neuropharmacology. 1996 Mar;35(3):267-72
pubmed: 8783200

Auteurs

Per Stenkrona (P)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm County Council, Stockholm, Sweden.

Granville J Matheson (GJ)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm County Council, Stockholm, Sweden.

Christer Halldin (C)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm County Council, Stockholm, Sweden.

Simon Cervenka (S)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm County Council, Stockholm, Sweden.

Lars Farde (L)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm County Council, Stockholm, Sweden.
PET Science Centre, Precision Medicine, R&D Oncology, AstraZeneca, Karolinska Institutet, Sweden.

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Classifications MeSH