Low eicosapentaenoic acid and gamma-linolenic acid levels in breast adipose tissue are associated with inflammatory breast cancer.


Journal

Breast (Edinburgh, Scotland)
ISSN: 1532-3080
Titre abrégé: Breast
Pays: Netherlands
ID NLM: 9213011

Informations de publication

Date de publication:
Jun 2019
Historique:
received: 06 09 2018
revised: 24 02 2019
accepted: 01 04 2019
pubmed: 9 4 2019
medline: 4 12 2019
entrez: 9 4 2019
Statut: ppublish

Résumé

Since it is thought that breast adipose tissue could influence breast cancer clinical presentation, we wanted to characterize specifically the relationship between breast adipose tissue fatty acid profile and Inflammatory Breast cancer (IBC). Two hundred thirty-four women presenting with breast cancer were managed in our centre between January 2009 and December 2011. Breast adipose tissue specimens were collected during breast surgery. We established the biochemical profile of adipose tissue fatty acids (FA) by gas chromatography and assessed whether there were differences in function of the presence of breast inflammation or not. We found that IBC was associated with decreased levels in breast adipose tissue of eicosapentaenoic acid (EPA), one of the two main polyunsaturated n-3 fatty acids (n-3 PUFA) of marine origin, but also with decreased levels of Gamma Linolenic acid (GLA). Inversely, an increase in palmitic acid levels was associated with IBC. These differences in lipid content may contribute to the occurrence of breast cancer inflammation.

Identifiants

pubmed: 30959386
pii: S0960-9776(19)30481-3
doi: 10.1016/j.breast.2019.04.001
pii:
doi:

Substances chimiques

gamma-Linolenic Acid 78YC2MAX4O
Eicosapentaenoic Acid AAN7QOV9EA

Types de publication

Evaluation Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

113-117

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Marie Chas (M)

Department of Gynecology, Centre Hospitalier Régional Universitaire de Tours, Hôpital Bretonneau. 2 Boulevard Tonnellé, 37044, Tours, France; INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France.

Caroline Goupille (C)

INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France.

Flavie Arbion (F)

Department of Pathology, Centre Hospitalier Régional Universitaire de Tours. Hôpital Bretonneau, 2 Boulevard Tonnellé, Tours, France.

Philippe Bougnoux (P)

INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France; Department of Oncology, Centre Hospitalier Régional Universitaire de Tours. Hôpital Bretonneau, 2 Boulevard Tonnellé, Tours, France.

Michelle Pinault (M)

INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France.

Marie Lise Jourdan (ML)

INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France.

Stephan Chevalier (S)

INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France.

Lobna Ouldamer (L)

Department of Gynecology, Centre Hospitalier Régional Universitaire de Tours, Hôpital Bretonneau. 2 Boulevard Tonnellé, 37044, Tours, France; INSERM UMR1069 « Nutrition, Growth and Cancer », 10 Boulevard Tonnellé, 37032, Tours, France; University of Tours, 10 Boulevard Tonnellé, 37032, Tours, France. Electronic address: l.ouldamer@chu-tours.fr.

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Classifications MeSH