The impact of bariatric surgery on asthma control differs among obese individuals with reported prior or current asthma, with or without metabolic syndrome.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2019
Historique:
received: 30 10 2018
accepted: 19 03 2019
entrez: 10 4 2019
pubmed: 10 4 2019
medline: 20 12 2019
Statut: epublish

Résumé

Both obesity and the metabolic syndrome have been independently associated with increased asthma morbidity. However, it is unclear whether metabolic syndrome limits the beneficial effects of weight loss on asthma. To evaluate whether bariatric weight loss is associated with improved asthma control, and whether this association varies by metabolic syndrome status. We determined the changes in asthma control, defined by the Asthma Control Test (ACT), before and after bariatric surgery among participants with asthma in the multi-center Longitudinal Assessment of Bariatric Surgery (LABS) study, stratifying our analysis by the presence or absence of metabolic syndrome. Among 2,458 LABS participants, 555 participants had an asthma diagnosis and were included in our analysis. Of these, 78% (n = 433) met criteria for metabolic syndrome (MetSyn) at baseline. In patients without MetSyn, mean ACT increased from 20.4 at baseline to 22.1 by 12-24 months, ending at 21.3 at 60 months. In contrast, among those with MetSyn there was no significant improvement in ACT scores. The proportion of patients without MetSyn with adequate asthma control (ACT >19) increased from 58% at baseline to 78% and 82% at 12 and 60 months, respectively, whereas among those with MetSyn, it was 73.8% at baseline, 77.1% at 12 months, dropping to 47.1% at 60 months (p = 0.004 for interaction between metabolic syndrome and time). Having MetSyn also increased the likelihood of losing asthma control during follow-up (HR = 1.92, 95% confidence interval [CI] 1.24-2.97, p = 0.003). Metabolic syndrome may negatively modify the effect of bariatric surgery-induced weight loss on asthma control.

Sections du résumé

BACKGROUND
Both obesity and the metabolic syndrome have been independently associated with increased asthma morbidity. However, it is unclear whether metabolic syndrome limits the beneficial effects of weight loss on asthma.
OBJECTIVES
To evaluate whether bariatric weight loss is associated with improved asthma control, and whether this association varies by metabolic syndrome status.
METHODS
We determined the changes in asthma control, defined by the Asthma Control Test (ACT), before and after bariatric surgery among participants with asthma in the multi-center Longitudinal Assessment of Bariatric Surgery (LABS) study, stratifying our analysis by the presence or absence of metabolic syndrome.
RESULTS
Among 2,458 LABS participants, 555 participants had an asthma diagnosis and were included in our analysis. Of these, 78% (n = 433) met criteria for metabolic syndrome (MetSyn) at baseline. In patients without MetSyn, mean ACT increased from 20.4 at baseline to 22.1 by 12-24 months, ending at 21.3 at 60 months. In contrast, among those with MetSyn there was no significant improvement in ACT scores. The proportion of patients without MetSyn with adequate asthma control (ACT >19) increased from 58% at baseline to 78% and 82% at 12 and 60 months, respectively, whereas among those with MetSyn, it was 73.8% at baseline, 77.1% at 12 months, dropping to 47.1% at 60 months (p = 0.004 for interaction between metabolic syndrome and time). Having MetSyn also increased the likelihood of losing asthma control during follow-up (HR = 1.92, 95% confidence interval [CI] 1.24-2.97, p = 0.003).
CONCLUSIONS
Metabolic syndrome may negatively modify the effect of bariatric surgery-induced weight loss on asthma control.

Identifiants

pubmed: 30964910
doi: 10.1371/journal.pone.0214730
pii: PONE-D-18-29872
pmc: PMC6456172
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0214730

Subventions

Organisme : NIDDK NIH HHS
ID : U01 DK066557
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK066667
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK066568
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK066471
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK066526
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK066555
Pays : United States

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Erick Forno (E)

Department of Pediatrics University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.

Peng Zhang (P)

Department of Medicine University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.

Mehdi Nouraie (M)

Department of Medicine University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.

Anita Courcoulas (A)

Department of Surgery University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.

James E Mitchell (JE)

Neutropsychiatric Research Institute, Fargo, North Dakota, United States of America.

Bruce M Wolfe (BM)

Dept. of Surgery, Oregon Health and Science University, Portland, Oregon, United States of America.

Gladys Strain (G)

Dept. of Surgery, Weill Cornell Medical College, New York, New York, United States of America.

Saurabh Khandelwal (S)

Dept. of Surgery, University of Washington Medical Center, Seattle, Washington, United States of America.

Fernando Holguin (F)

Dept. of Medicine, University of Colorado, Denver, Colorado, United States of America.

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Classifications MeSH