Ki-67 is an independent predictor of prostate cancer death in routine needle biopsy samples: proving utility for routine assessments.


Journal

Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc
ISSN: 1530-0285
Titre abrégé: Mod Pathol
Pays: United States
ID NLM: 8806605

Informations de publication

Date de publication:
09 2019
Historique:
received: 14 11 2018
accepted: 12 03 2019
revised: 06 03 2019
pubmed: 13 4 2019
medline: 7 7 2020
entrez: 13 4 2019
Statut: ppublish

Résumé

Standard clinical parameters fail to accurately differentiate indolent from aggressive prostate cancer. Our previous studies showed that immunohistochemical testing for Ki-67 improved prediction of prostate cancer death in a previous cohort of conservatively treated clinically localized prostate cancer. However there is a need for validation of usage with whole biopsy sections rather than tissue micro-arrays for use in routine diagnostics. Prostate cancer biopsy cases were identified in the UK, between 1990 and 2003, treated conservatively. Tumor extent and prostate-specific antigen (PSA) serum measurements were available. Biopsy cases were centrally reviewed by three uropathologists and Gleason conformed to contemporary ISUP 2014 criteria. Follow-up was through cancer registries up until 2012. Deaths were divided into those from prostate cancer and those from other causes. The percentage of Ki-67 in tumor cells was evaluated by immunohistochemistry on whole biopsy sections and was available for 756 patients. This percentage was used in analysis of cancer specific survival using a Cox proportional hazards model. In univariate analysis, the interquartile hazard ratio (HR) (95% confidence intervals) for continuous Ki-67 was 1.68 (1.49, 1.89), χ

Identifiants

pubmed: 30976102
doi: 10.1038/s41379-019-0268-y
pii: S0893-3952(22)01017-1
pmc: PMC8647491
mid: NIHMS1759524
doi:

Substances chimiques

Biomarkers, Tumor 0
Ki-67 Antigen 0
MKI67 protein, human 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1303-1309

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA092629
Pays : United States
Organisme : NCI NIH HHS
ID : T32 CA009629
Pays : United States

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Auteurs

Solène-Florence Kammerer-Jacquet (SF)

Department of Molecular Oncology, Barts Cancer Institute, Queen Mary University of London, London, EC1A 7BE, UK. jacquet.sf@gmail.com.
Department of Pathology, University Hospital of Rennes, Université de Rennes 1, Université Bretagne Loire, 35000, Rennes, France. jacquet.sf@gmail.com.

Amar Ahmad (A)

UK Centre for Cancer Prevention, Wolfson Institute of Preventive Medicine, Queen Mary University of London, London, EC1A 7BE, UK.

Henrik Møller (H)

Cancer Epidemiology and Population Health, King's College London, London, SE1 9RT, UK.

Holly Sandu (H)

UK Centre for Cancer Prevention, Wolfson Institute of Preventive Medicine, Queen Mary University of London, London, EC1A 7BE, UK.

Peter Scardino (P)

Department of Urology, Memorial Sloan-Kettering Cancer Center, New York, NY, 10065 NY, USA.

Geraldine Soosay (G)

Department of Pathology, Queen's Hospital, RM7 0AG, Romford, Essex, UK.

Luis Beltran (L)

Department of Molecular Oncology, Barts Cancer Institute, Queen Mary University of London, London, EC1A 7BE, UK.

Jack Cuzick (J)

UK Centre for Cancer Prevention, Wolfson Institute of Preventive Medicine, Queen Mary University of London, London, EC1A 7BE, UK.

Daniel M Berney (DM)

Department of Molecular Oncology, Barts Cancer Institute, Queen Mary University of London, London, EC1A 7BE, UK.

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Classifications MeSH