Cerebellar transcranial direct current stimulation modulates the fMRI signal in the cerebellar nuclei in a simple motor task.


Journal

Brain stimulation
ISSN: 1876-4754
Titre abrégé: Brain Stimul
Pays: United States
ID NLM: 101465726

Informations de publication

Date de publication:
Historique:
received: 12 08 2016
revised: 31 03 2019
accepted: 01 04 2019
pubmed: 17 4 2019
medline: 14 1 2020
entrez: 17 4 2019
Statut: ppublish

Résumé

In a seminal paper, Galea et al. (Modulation of cerebellar excitability by polarity-specific noninvasive direct current stimulation. 2009. J Neurosci 29, 9115-9122) showed that cerebellar transcranial direct current stimulation (ctDCS) alters cerebellar-M1 connectivity. This effect has been explained by ctDCS-related changes of excitability of the cerebellar cortex with consecutive modulation of its main output, the dentate-thalamo-cortical pathway. The aim of this functional magnetic resonance imaging (fMRI) study was to provide evidence that cathodal ctDCS decreases the activity of the cerebellar cortex, resulting in increased activity of the cerebellar nuclei, whereas anodal ctDCS has the opposite effect. A total of 48 participants (female/male: 23/25, age: 23.8 ± 4.1yrs., mean ± standard deviation) performed a finger tapping task with the right hand in a 3T MRI scanner. Functional MR images were acquired prior, during and after tDCS of the right cerebellum. Participants were assigned randomly to anodal, cathodal or sham ctDCS. No significant difference of cerebellar cortical activation was found after comparing the three modes of stimulation. On the level of the dentate nuclei, however, a significant increase of activation was detected during and after cathodal stimulation. Furthermore, dentate nuclei activation was suppressed on a trend level following anodal stimulation. The present findings support the hypothesis that cathodal ctDCS leads to a disinhibition of the dentate nucleus, whereas anodal ctDCS may have the opposite effect.

Sections du résumé

BACKGROUND
In a seminal paper, Galea et al. (Modulation of cerebellar excitability by polarity-specific noninvasive direct current stimulation. 2009. J Neurosci 29, 9115-9122) showed that cerebellar transcranial direct current stimulation (ctDCS) alters cerebellar-M1 connectivity. This effect has been explained by ctDCS-related changes of excitability of the cerebellar cortex with consecutive modulation of its main output, the dentate-thalamo-cortical pathway.
OBJECTIVES
The aim of this functional magnetic resonance imaging (fMRI) study was to provide evidence that cathodal ctDCS decreases the activity of the cerebellar cortex, resulting in increased activity of the cerebellar nuclei, whereas anodal ctDCS has the opposite effect.
METHODS
A total of 48 participants (female/male: 23/25, age: 23.8 ± 4.1yrs., mean ± standard deviation) performed a finger tapping task with the right hand in a 3T MRI scanner. Functional MR images were acquired prior, during and after tDCS of the right cerebellum. Participants were assigned randomly to anodal, cathodal or sham ctDCS.
RESULTS
No significant difference of cerebellar cortical activation was found after comparing the three modes of stimulation. On the level of the dentate nuclei, however, a significant increase of activation was detected during and after cathodal stimulation. Furthermore, dentate nuclei activation was suppressed on a trend level following anodal stimulation.
CONCLUSIONS
The present findings support the hypothesis that cathodal ctDCS leads to a disinhibition of the dentate nucleus, whereas anodal ctDCS may have the opposite effect.

Identifiants

pubmed: 30987860
pii: S1935-861X(19)30187-1
doi: 10.1016/j.brs.2019.04.002
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1169-1176

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Michael Küper (M)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany. Electronic address: Michael.Kueper@uni-due.de.

Jahan Saeed Mallick (JS)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

Thomas Ernst (T)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

Oliver Kraff (O)

Institute of Diagnostic and Interventional Radiology and Neuroradiology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany; Erwin L. Hahn Institute for Magnetic Resonance Imaging, Kokereiallee 7, Building C84 UNESCO World Heritage, Zeche Zollverein, 45141, Essen, Germany.

Markus Thürling (M)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

Maria Roxana Stefanescu (MR)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

Sophia Göricke (S)

Institute of Diagnostic and Interventional Radiology and Neuroradiology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

Michael A Nitsche (MA)

Department of Psychology and Neurosciences, Leibniz Research Centre for Working Environment and Human Factors, Ardeystr.67, 44139, Dortmund, Germany; Department of Neurology, University Medical Hospital Bergmannsheil, Bürkle de la Camp-Platz 1, 44789, Bochum, Germany.

Dagmar Timmann (D)

Department of Neurology, University of Duisburg-Essen, Hufelandstrasse 55, 45122, Essen, Germany.

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