Heterogeneous Expression of Programmed Death Receptor-ligand 1 on Circulating Tumor Cells in Patients With Lung Cancer.


Journal

Clinical lung cancer
ISSN: 1938-0690
Titre abrégé: Clin Lung Cancer
Pays: United States
ID NLM: 100893225

Informations de publication

Date de publication:
07 2019
Historique:
received: 08 01 2019
revised: 20 02 2019
accepted: 17 03 2019
pubmed: 22 4 2019
medline: 4 4 2020
entrez: 22 4 2019
Statut: ppublish

Résumé

Blockade of the programmed death receptor-1 (PD-1) pathway is effective against solid tumors including lung cancer. PD-ligand 1 (PD-L1) expression on tumor tissue serves as a predictive biomarker for the efficacy of PD-1 pathway blockade. Here, we evaluated the expression of PD-L1 on circulating tumor cells (CTCs) in patients with lung cancer. Peripheral whole blood (3 mL) was collected from patients, and CTCs and PD-L1 expression were detected using a microcavity array (MCA) system. Immunohistochemistry for PD-L1 detection was also performed using matched tumor tissues. Sixty-seven patients with lung cancer were enrolled in the study between July 2015 and April 2016 at Wakayama Medical University Hospital. The characteristics of the patients were as follows: median age, 71 years (range, 39-86 years); male, 72%; stage II to III/IV, 14%/85%; non-small-cell lung cancer/small-cell lung cancer/other, 73%/21%/6%. CTCs were detected in 66 of 67 patients (median, 19; range, 0-115), and more than 5 CTCs were detected in 78% of patients. PD-L1-expressing CTCs were detected in 73% of patients, and the proportion score of PD-L1-expressing CTCs ranged from 3% to 100%, suggesting intra-patient heterogeneity of PD-L1 expression on CTCs. Tumor tissues were available from 27 patients and were immunostained for PD-L1, and no correlation was observed between tumor tissues and CTCs based on the proportion score (R PD-L1 expression was detectable on CTCs in patients with lung cancer, and intra-patient heterogeneity was observed. No correlation was observed between PD-L1 expression in tumor tissues and CTCs.

Sections du résumé

BACKGROUND
Blockade of the programmed death receptor-1 (PD-1) pathway is effective against solid tumors including lung cancer. PD-ligand 1 (PD-L1) expression on tumor tissue serves as a predictive biomarker for the efficacy of PD-1 pathway blockade. Here, we evaluated the expression of PD-L1 on circulating tumor cells (CTCs) in patients with lung cancer.
MATERIALS AND METHODS
Peripheral whole blood (3 mL) was collected from patients, and CTCs and PD-L1 expression were detected using a microcavity array (MCA) system. Immunohistochemistry for PD-L1 detection was also performed using matched tumor tissues.
RESULTS
Sixty-seven patients with lung cancer were enrolled in the study between July 2015 and April 2016 at Wakayama Medical University Hospital. The characteristics of the patients were as follows: median age, 71 years (range, 39-86 years); male, 72%; stage II to III/IV, 14%/85%; non-small-cell lung cancer/small-cell lung cancer/other, 73%/21%/6%. CTCs were detected in 66 of 67 patients (median, 19; range, 0-115), and more than 5 CTCs were detected in 78% of patients. PD-L1-expressing CTCs were detected in 73% of patients, and the proportion score of PD-L1-expressing CTCs ranged from 3% to 100%, suggesting intra-patient heterogeneity of PD-L1 expression on CTCs. Tumor tissues were available from 27 patients and were immunostained for PD-L1, and no correlation was observed between tumor tissues and CTCs based on the proportion score (R
CONCLUSION
PD-L1 expression was detectable on CTCs in patients with lung cancer, and intra-patient heterogeneity was observed. No correlation was observed between PD-L1 expression in tumor tissues and CTCs.

Identifiants

pubmed: 31005568
pii: S1525-7304(19)30077-4
doi: 10.1016/j.cllc.2019.03.004
pii:
doi:

Substances chimiques

Antineoplastic Agents, Immunological 0
B7-H1 Antigen 0
Biomarkers, Pharmacological 0
CD274 protein, human 0
PDCD1 protein, human 0
Programmed Cell Death 1 Receptor 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

270-277.e1

Informations de copyright

Copyright © 2019 The Author(s). Published by Elsevier Inc. All rights reserved.

Auteurs

Yasuhiro Koh (Y)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan. Electronic address: ykoh@wakayama-med.ac.jp.

Satomi Yagi (S)

Medical Business Unit, Hitachi Chemical Co, Ltd, Ibaraki, Japan.

Hiroaki Akamatsu (H)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Kuninobu Kanai (K)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Atsushi Hayata (A)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Nahomi Tokudome (N)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Keiichiro Akamatsu (K)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Masayuki Higuchi (M)

Medical Business Unit, Hitachi Chemical Co, Ltd, Ibaraki, Japan.

Hisashige Kanbara (H)

Medical Business Unit, Hitachi Chemical Co, Ltd, Ibaraki, Japan.

Masanori Nakanishi (M)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Hiroki Ueda (H)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

Nobuyuki Yamamoto (N)

Internal Medicine III, Wakayama Medical University, Wakayama, Japan.

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Classifications MeSH