Role of Perivascular Oligodendrocyte Precursor Cells in Angiogenesis After Brain Ischemia.
Angiogenic Proteins
/ genetics
Animals
Behavior, Animal
Brain
/ blood supply
Cell Hypoxia
Cells, Cultured
Culture Media, Conditioned
/ metabolism
Disease Models, Animal
Endothelial Cells
/ metabolism
Infarction, Middle Cerebral Artery
/ metabolism
Male
Mice, Inbred C57BL
Motor Activity
Neovascularization, Physiologic
Oligodendroglia
/ metabolism
Paracrine Communication
Phenotype
Rats, Sprague-Dawley
Recovery of Function
Signal Transduction
Stem Cells
/ metabolism
angiogenesis
glial cell
stem cell
stroke
Journal
Journal of the American Heart Association
ISSN: 2047-9980
Titre abrégé: J Am Heart Assoc
Pays: England
ID NLM: 101580524
Informations de publication
Date de publication:
07 05 2019
07 05 2019
Historique:
entrez:
26
4
2019
pubmed:
26
4
2019
medline:
4
8
2020
Statut:
ppublish
Résumé
Background Oligodendrocyte precursor cells ( OPC s) regulate neuronal, glial, and vascular systems in diverse ways and display phenotypic heterogeneity beyond their established role as a reservoir for mature oligodendrocytes. However, the detailed phenotypic changes of OPC s after cerebral ischemia remain largely unknown. Here, we aimed to investigate the roles of reactive OPC s in the ischemic brain. Methods and Results The behavior of OPC s was evaluated in a mouse model of ischemic stroke produced by transient middle cerebral artery occlusion in vivo. For in vitro experiments, the phenotypic change of OPC s after oxygen glucose derivation was examined using a primary rat OPC culture. Furthermore, the therapeutic potential of hypoxic OPC s was evaluated in a mouse model of middle cerebral artery occlusion in vivo. Perivascular OPC s in the cerebral cortex were increased alongside poststroke angiogenesis in a mouse model of middle cerebral artery occlusion. In vitro RNA -seq analysis revealed that primary cultured OPC s increased the gene expression of numerous pro-angiogenic factors after oxygen glucose derivation. Hypoxic OPC s secreted a greater amount of pro-angiogenic factors, such as vascular endothelial growth factor and angiopoietin-1, compared with normoxic OPC s. Hypoxic OPC -derived conditioned media increased the viability and tube formation of endothelial cells. In vivo studies also demonstrated that 5 consecutive daily treatments with hypoxic OPC -conditioned media, beginning 2 days after middle cerebral artery occlusion, facilitated poststroke angiogenesis, alleviated infarct volume, and improved functional disabilities. Conclusions Following cerebral ischemia, the phenotype of OPC s in the cerebral cortex shifts from the parenchymal subtype to the perivascular subtype, which can promote angiogenesis. The optimal use of hypoxic OPC s secretome would provide a novel therapeutic option for stroke.
Identifiants
pubmed: 31020902
doi: 10.1161/JAHA.118.011824
pmc: PMC6512138
doi:
Substances chimiques
Angiogenic Proteins
0
Culture Media, Conditioned
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
e011824Subventions
Organisme : NINDS NIH HHS
ID : R01 NS065089
Pays : United States
Organisme : NINDS NIH HHS
ID : P01 NS055104
Pays : United States
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