In Vitro Grown Micro-Tissues for Cardiac Cell Replacement Therapy in Vivo.


Journal

Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology
ISSN: 1421-9778
Titre abrégé: Cell Physiol Biochem
Pays: Germany
ID NLM: 9113221

Informations de publication

Date de publication:
2019
Historique:
received: 14 09 2018
accepted: 29 04 2019
entrez: 4 5 2019
pubmed: 3 5 2019
medline: 15 5 2019
Statut: ppublish

Résumé

Different approaches have been considered to improve heart reconstructive medicine and direct delivery of pluripotent stem cell-derived cardiomyocytes (PSC-CMs) appears to be highly promising in this context. However, low cell persistence post-transplantation remains a bottleneck hindering the approach. Here, we present a novel strategy to overcome the low engraftment of PSC-CMs during the early post-transplantation phase into the myocardium of both healthy and cryoinjured syngeneic mice. Adult murine bone marrow mesenchymal stem cells (MSCs) and PSC-CMs were co-cultured on thermo-responsive polymers and later detached through temperature reduction, resulting in the protease-free generation of cell clusters (micro-tissues) composed of both cells types. Micro-tissues were transplanted into healthy and cryo-injured murine hearts. Short term cell retention was quantified by real-time-PCR. Longitudinal cell tracking was performed by bioluminescence imaging for four weeks. Transplanted cells were further detected by immunofluorescence staining of tissue sections. We demonstrated that in vitro grown micro-tissues consisting of PSC-CMs and MSCs can increase cardiomyocyte retention by >10fold one day post-transplantation, but could not fully rescue a further cell loss between day 1 and day 2. Neutrophil infiltration into the transplanted area was detected in healthy hearts and could be attributed to the cellular implantation rather than tissue damage exerted by the transplantation cannula. Injected PSC-CMs were tracked and successfully detected for up to four weeks by bioluminescence imaging. This approach demonstrated that in vitro grown micro-tissues might contribute to the development of cardiac cell replacement therapies.

Sections du résumé

BACKGROUND/AIMS OBJECTIVE
Different approaches have been considered to improve heart reconstructive medicine and direct delivery of pluripotent stem cell-derived cardiomyocytes (PSC-CMs) appears to be highly promising in this context. However, low cell persistence post-transplantation remains a bottleneck hindering the approach. Here, we present a novel strategy to overcome the low engraftment of PSC-CMs during the early post-transplantation phase into the myocardium of both healthy and cryoinjured syngeneic mice.
METHODS METHODS
Adult murine bone marrow mesenchymal stem cells (MSCs) and PSC-CMs were co-cultured on thermo-responsive polymers and later detached through temperature reduction, resulting in the protease-free generation of cell clusters (micro-tissues) composed of both cells types. Micro-tissues were transplanted into healthy and cryo-injured murine hearts. Short term cell retention was quantified by real-time-PCR. Longitudinal cell tracking was performed by bioluminescence imaging for four weeks. Transplanted cells were further detected by immunofluorescence staining of tissue sections.
RESULTS RESULTS
We demonstrated that in vitro grown micro-tissues consisting of PSC-CMs and MSCs can increase cardiomyocyte retention by >10fold one day post-transplantation, but could not fully rescue a further cell loss between day 1 and day 2. Neutrophil infiltration into the transplanted area was detected in healthy hearts and could be attributed to the cellular implantation rather than tissue damage exerted by the transplantation cannula. Injected PSC-CMs were tracked and successfully detected for up to four weeks by bioluminescence imaging.
CONCLUSION CONCLUSIONS
This approach demonstrated that in vitro grown micro-tissues might contribute to the development of cardiac cell replacement therapies.

Identifiants

pubmed: 31050280
doi: 10.33594/000000092
doi:

Substances chimiques

Polymers 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1309-1324

Subventions

Organisme : Else Kröner-Fresenius-Stiftung
ID : 2012_A143
Pays : Germany

Informations de copyright

© Copyright by the Author(s). Published by Cell Physiol Biochem Press.

Déclaration de conflit d'intérêts

The authors declare to have no competing interests.

Auteurs

Raja Ghazanfar Ali Sahito (RGA)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Xiaowu Sheng (X)

Hunan Cancer Hospital and Affiliated Cancer Hospital of Xiangya School of Medicine, Central South University, Changsha, China.

Martina Maass (M)

Department III of Internal Medicine, University Hospital of Cologne, Cologne, Germany.

Nelly Mikhael (N)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Sarkawt Hamad (S)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Carlos O Heras-Bautista (CO)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Daniel Derichsweiler (D)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Dimitry Spitkovsky (D)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Frank Suhr (F)

Exercise Physiology Research Group, Department of Movement Sciences, Group of Biomedical Sciences, KU Leuven, Leuven, Belgium.

Markus Khalil (M)

Department for Pediatric Cardiology, University of Giessen, Giessen, Germany.

Konrad Brockmeier (K)

Department for Pediatric Cardiology, University Hospital of Cologne, Cologne, Germany.

Marcel Halbach (M)

Department III of Internal Medicine, University Hospital of Cologne, Cologne, Germany.

Tomo Saric (T)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Jürgen Hescheler (J)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.

Benjamin Krausgrill (B)

Department III of Internal Medicine, University Hospital of Cologne, Cologne, Germany, benjamin.krausgrill@uk-koeln.de.

Kurt Pfannkuche (K)

Center for Physiology and Pathophysiology, Institute of Neurophysiology, Medical Faculty, University of Cologne, Cologne, Germany.
Department for Pediatric Cardiology, University Hospital of Cologne, Cologne, Germany, kurt.pfannkuche@uni-koeln.de.

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