Dopaminergic Vulnerability in Parkinson Disease: The Cost of Humans' Habitual Performance.

Parkinson disease dopamine goal-directed behavior habitual behavior vulnerability

Journal

Trends in neurosciences
ISSN: 1878-108X
Titre abrégé: Trends Neurosci
Pays: England
ID NLM: 7808616

Informations de publication

Date de publication:
06 2019
Historique:
received: 12 10 2018
revised: 21 01 2019
accepted: 19 03 2019
pubmed: 6 5 2019
medline: 22 9 2020
entrez: 5 5 2019
Statut: ppublish

Résumé

Humans can simultaneously combine automatic/habitual and voluntary/goal-directed aspects of behavioral control. Habitual routines permit us to perform well practiced task-components with minimal or no voluntary attention. Evidence from animal and human investigations indicates that dopaminergic neurons in lateral substantia nigra, which innervate the sensorimotor striatum, are engaged during the acquisition and performance of automatized skills and habits. Typically, in Parkinson disease (PD), there is a differential loss of dopamine, which occurs earliest and most severely in the caudal sensorimotor striatum, a subdivision of the striatum implicated in habitual control. We suggest that frequent reliance on habitual performance may be a critical functional stressor, which, when combined with other more general risk factors, could explain the selective neurodegeneration of the nigrostriatal motor projection in PD.

Identifiants

pubmed: 31053241
pii: S0166-2236(19)30041-4
doi: 10.1016/j.tins.2019.03.007
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

375-383

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Ledia F Hernandez (LF)

HM CINAC, Hospital Universitario HM Puerta del Sur, Mostoles, Madrid, Spain; Universidad CEU San Pablo, Madrid, Spain; Center for Networked Biomedical Research on Neurodegenerative Diseases (CIBERNED), Madrid, Spain.

Ignacio Obeso (I)

HM CINAC, Hospital Universitario HM Puerta del Sur, Mostoles, Madrid, Spain; Universidad CEU San Pablo, Madrid, Spain; Center for Networked Biomedical Research on Neurodegenerative Diseases (CIBERNED), Madrid, Spain.

Rui M Costa (RM)

Champalimaud Neuroscience Programme, Champalimaud Centre for the Unknown, Lisbon, Portugal; Departments of Neuroscience and Neurology, Zuckerman Mind Brain Behavior Institute, Columbia University, New York, NY, USA.

Peter Redgrave (P)

Department of Psychology, University of Sheffield, Sheffield, S10 2TN, UK.

Jose A Obeso (JA)

HM CINAC, Hospital Universitario HM Puerta del Sur, Mostoles, Madrid, Spain; Universidad CEU San Pablo, Madrid, Spain; Center for Networked Biomedical Research on Neurodegenerative Diseases (CIBERNED), Madrid, Spain. Electronic address: jobeso.hmcinac@hmhospitales.com.

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Classifications MeSH