Circulating transforming growth factor-β1 facilitates remyelination in the adult central nervous system.


Journal

eLife
ISSN: 2050-084X
Titre abrégé: Elife
Pays: England
ID NLM: 101579614

Informations de publication

Date de publication:
09 05 2019
Historique:
received: 10 09 2018
accepted: 26 04 2019
entrez: 10 5 2019
pubmed: 10 5 2019
medline: 26 2 2020
Statut: epublish

Résumé

Oligodendrocyte maturation is necessary for functional regeneration in the CNS; however, the mechanisms by which the systemic environment regulates oligodendrocyte maturation is unclear. We found that Transforming growth factor (TGF)-β1, which is present in higher levels in the systemic environment, promotes oligodendrocyte maturation. Oligodendrocyte maturation was enhanced by adult mouse serum treatment via TGF-β type I receptor. Decrease in circulating TGF-β1 level prevented remyelination in the spinal cord after toxin-induced demyelination. TGF-β1 administration promoted remyelination and restored neurological function in a multiple sclerosis animal model. Furthermore, TGF-β1 treatment stimulated human oligodendrocyte maturation. These data provide the therapeutic possibility of TGF-β for demyelinating diseases.

Identifiants

pubmed: 31071011
doi: 10.7554/eLife.41869
pii: 41869
pmc: PMC6508935
doi:
pii:

Substances chimiques

Transforming Growth Factor beta1 0

Banques de données

Dryad
['10.5061/dryad.nj51t60']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Japan Society for the Promotion of Science
ID : 16H04672
Pays : International
Organisme : The Uehara Memorial Foundation
ID : 2017
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 17H06178
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 19H03554
Pays : International

Informations de copyright

© 2019, Hamaguchi et al.

Déclaration de conflit d'intérêts

MH, RM, HF, HM, HK, TY No competing interests declared

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Auteurs

Machika Hamaguchi (M)

Department of Molecular Neuroscience, Graduate School of Medicine, Osaka University, Suita, Japan.

Rieko Muramatsu (R)

Department of Molecular Neuroscience, Graduate School of Medicine, Osaka University, Suita, Japan.
Department of Molecular Pharmacology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Japan.
WPI Immunology Frontier Research Center, Osaka University, Suita, Japan.

Harutoshi Fujimura (H)

Toneyama National Hospital, Toyonaka, Japan.

Hideki Mochizuki (H)

Department of Neurology, Graduate School of Medicine, Osaka University, Suita, Japan.

Hirotoshi Kataoka (H)

Department of Neuro-Medical Science, Graduate School of Medicine, Osaka University, Suita, Japan.

Toshihide Yamashita (T)

Department of Molecular Neuroscience, Graduate School of Medicine, Osaka University, Suita, Japan.
WPI Immunology Frontier Research Center, Osaka University, Suita, Japan.
Department of Neuro-Medical Science, Graduate School of Medicine, Osaka University, Suita, Japan.
Graduate School of Frontier Biosciences, Osaka University, Suita, Japan.

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