Platelet Depletion/Transfusion as a Lethal Factor in a Colitis-associated Cancer Mouse Model.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
May 2019
Historique:
received: 07 03 2019
revised: 30 03 2019
accepted: 03 04 2019
entrez: 17 5 2019
pubmed: 17 5 2019
medline: 23 5 2019
Statut: ppublish

Résumé

TLR-4 Knock-out (KO) mice are protected from colitis-associated cancer in the established AOM/DSS mouse model. The aim of this study was to assess whether the TLR4 KO mice would still be protected from carcinogenesis after platelet depletion and transfusion with TLR4 wild-type platelets. Thirty-two female C57BL6 mice were divided into 6 groups. Among the three groups that received Azoxymethane/Dextran Sulfate Sodium (AOM/DSS), one group included TLR4KO mice, which were depleted of their platelets and were then transfused with platelets from TLR4 wild-type mice. The other two groups included wild-type and TLR-4KO mice that only received AOM/DSS. All 6 animals in the KO group that underwent platelet depletion/transfusion succumbed. Three of them died before the administration of DSS and three in the week following DSS administration. In contrast, mice in the other two groups experienced less weight loss and only 1 mouse died in each of them. Platelet depletion/transfusion was detrimental in TLR-4 transgenic mice that received AOM/DSS.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
TLR-4 Knock-out (KO) mice are protected from colitis-associated cancer in the established AOM/DSS mouse model. The aim of this study was to assess whether the TLR4 KO mice would still be protected from carcinogenesis after platelet depletion and transfusion with TLR4 wild-type platelets.
MATERIALS AND METHODS METHODS
Thirty-two female C57BL6 mice were divided into 6 groups. Among the three groups that received Azoxymethane/Dextran Sulfate Sodium (AOM/DSS), one group included TLR4KO mice, which were depleted of their platelets and were then transfused with platelets from TLR4 wild-type mice. The other two groups included wild-type and TLR-4KO mice that only received AOM/DSS.
RESULTS RESULTS
All 6 animals in the KO group that underwent platelet depletion/transfusion succumbed. Three of them died before the administration of DSS and three in the week following DSS administration. In contrast, mice in the other two groups experienced less weight loss and only 1 mouse died in each of them.
CONCLUSION CONCLUSIONS
Platelet depletion/transfusion was detrimental in TLR-4 transgenic mice that received AOM/DSS.

Identifiants

pubmed: 31092437
pii: 39/5/2443
doi: 10.21873/anticanres.13362
doi:

Substances chimiques

Tlr4 protein, mouse 0
Toll-Like Receptor 4 0
Dextran Sulfate 9042-14-2
Azoxymethane MO0N1J0SEN

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2443-2446

Informations de copyright

Copyright© 2019, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Anastasios Angelou (A)

Second Department of Propedeutic Surgery, Laiko General Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Efstathios Antoniou (E)

Second Department of Propedeutic Surgery, Laiko General Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Anastasia Pikouli (A)

3rd Department of Surgery, School of Medicine, National and Kapodistrian University of Athens, "Attikon" University General Hospital, Athens, Greece.

Jane Wang (J)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, MD, U.S.A.

Marco Ventin (M)

Department of Surgery, University of Udine, Udine, Italy.

Stefan Buettner (S)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, MD, U.S.A.

Muhammad Faateh (M)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, MD, U.S.A.

George Theodoropoulos (G)

First Department of Propaedeutic Surgery, Hippokratio General Hospital of Athens, Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Georgios C Zografos (GC)

First Department of Propaedeutic Surgery, Hippokratio General Hospital of Athens, Medical School, National and Kapodistrian University of Athens, Athens, Greece.

Stamatios Theocharis (S)

First Department of Pathology, National and Kapodistrian University of Athens, Athens, Greece.

Apostolos E Papalois (AE)

Experimental, Educational and Research Centre ELPEN, Athens, Greece.
Medical School, European University Cyprus, Nicosia, Cyprus.

Emmanouil Pikoulis (E)

3rd Department of Surgery, School of Medicine, National and Kapodistrian University of Athens, "Attikon" University General Hospital, Athens, Greece.

Georgios Antonios Margonis (GA)

Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, MD, U.S.A. antonis.margonis@gmail.com.

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Classifications MeSH