The Modulation of Neural Noise Underlies the Effectiveness of Methylphenidate Treatment in Attention-Deficit/Hyperactivity Disorder.


Journal

Biological psychiatry. Cognitive neuroscience and neuroimaging
ISSN: 2451-9030
Titre abrégé: Biol Psychiatry Cogn Neurosci Neuroimaging
Pays: United States
ID NLM: 101671285

Informations de publication

Date de publication:
08 2019
Historique:
received: 20 12 2018
revised: 19 03 2019
accepted: 26 03 2019
pubmed: 20 5 2019
medline: 28 3 2020
entrez: 20 5 2019
Statut: ppublish

Résumé

Various lines of research suggest that the stability of neural processes is low in attention-deficit/hyperactivity disorder (ADHD). Considering overarching neural principles, this lack of stability relates to increased levels of neural noise. However, no study has directly examined neural noise in ADHD. Likewise, it is unknown whether the modulation of neural noise reflects a mechanistic link as to why methylphenidate (MPH) is effective in treating impulsivity in ADHD. We compared neural noise between 29 juvenile patients with ADHD and 32 healthy control subjects and examined the effects of MPH. We examined 1/f neural noise of electroencephalogram data collected while participants performed a response inhibition (Go/NoGo) task. Specific during NoGo trials, children with ADHD showed more neural noise than healthy control subjects. This was especially the case with regard to the theta frequency band, which is very closely related to cognitive control. MPH treatment reduced neural noise in ADHD to the level of healthy control subjects. Correlational analyses showed a direct relationship between decreases in neural noise and increases in behavioral performance. Mechanistically, this can be explained by the MPH-induced increase in dopaminergic neurotransmission that enhances the signal-to-noise ratio in neural networks and thus reduces neural noise. This study is the first to demonstrate increased (pink) neural noise in patients with ADHD and its reduction through MPH treatment. The study reveals an important mechanistic link as to why MPH is effective in treating impulsivity in ADHD.

Sections du résumé

BACKGROUND
Various lines of research suggest that the stability of neural processes is low in attention-deficit/hyperactivity disorder (ADHD). Considering overarching neural principles, this lack of stability relates to increased levels of neural noise. However, no study has directly examined neural noise in ADHD. Likewise, it is unknown whether the modulation of neural noise reflects a mechanistic link as to why methylphenidate (MPH) is effective in treating impulsivity in ADHD.
METHODS
We compared neural noise between 29 juvenile patients with ADHD and 32 healthy control subjects and examined the effects of MPH. We examined 1/f neural noise of electroencephalogram data collected while participants performed a response inhibition (Go/NoGo) task.
RESULTS
Specific during NoGo trials, children with ADHD showed more neural noise than healthy control subjects. This was especially the case with regard to the theta frequency band, which is very closely related to cognitive control. MPH treatment reduced neural noise in ADHD to the level of healthy control subjects. Correlational analyses showed a direct relationship between decreases in neural noise and increases in behavioral performance. Mechanistically, this can be explained by the MPH-induced increase in dopaminergic neurotransmission that enhances the signal-to-noise ratio in neural networks and thus reduces neural noise.
CONCLUSIONS
This study is the first to demonstrate increased (pink) neural noise in patients with ADHD and its reduction through MPH treatment. The study reveals an important mechanistic link as to why MPH is effective in treating impulsivity in ADHD.

Identifiants

pubmed: 31103546
pii: S2451-9022(19)30080-1
doi: 10.1016/j.bpsc.2019.03.011
pii:
doi:

Substances chimiques

Central Nervous System Stimulants 0
Methylphenidate 207ZZ9QZ49

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

743-750

Informations de copyright

Copyright © 2019 Society of Biological Psychiatry. Published by Elsevier Inc. All rights reserved.

Auteurs

Maik Pertermann (M)

Cognitive Neurophysiology, Department of Child and Adolescent Psychiatry, Faculty of Medicine, Dresden University of Technology, Dresden, Germany.

Annet Bluschke (A)

Cognitive Neurophysiology, Department of Child and Adolescent Psychiatry, Faculty of Medicine, Dresden University of Technology, Dresden, Germany.

Veit Roessner (V)

Cognitive Neurophysiology, Department of Child and Adolescent Psychiatry, Faculty of Medicine, Dresden University of Technology, Dresden, Germany.

Christian Beste (C)

Cognitive Neurophysiology, Department of Child and Adolescent Psychiatry, Faculty of Medicine, Dresden University of Technology, Dresden, Germany. Electronic address: christian.beste@uniklinikum-dresden.de.

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Classifications MeSH