Sphingosine kinase 2 is a negative regulator of inflammatory macrophage activation.


Journal

Biochimica et biophysica acta. Molecular and cell biology of lipids
ISSN: 1879-2618
Titre abrégé: Biochim Biophys Acta Mol Cell Biol Lipids
Pays: Netherlands
ID NLM: 101731727

Informations de publication

Date de publication:
09 2019
Historique:
received: 20 09 2018
revised: 15 05 2019
accepted: 20 05 2019
pubmed: 28 5 2019
medline: 3 3 2020
entrez: 26 5 2019
Statut: ppublish

Résumé

Sphingosine kinases (SPHK) generate the sphingolipid sphingosine-1-phosphate, which, among other functions, is a potent regulator of inflammation. While SPHK1 produces S1P to promote inflammatory signaling, the role of SPHK2 is unclear due to divergent findings in studies utilizing gene depletion versus inhibition of catalytic activity. We sought to clarify how SPHK2 affects inflammatory signaling in human macrophages, which are main regulators of inflammation. SPHK2 expression and activity were rapidly decreased within 6 h upon stimulating primary human macrophages with lipopolysaccharide (LPS), but was upregulated after 24 h. At 24 h following LPS stimulation, targeting SPHK2 with the inhibitor ABC294640, a specific siRNA or by using Sphk2

Identifiants

pubmed: 31128248
pii: S1388-1981(18)30300-7
doi: 10.1016/j.bbalip.2019.05.008
pii:
doi:

Substances chimiques

Cytokines 0
Phosphotransferases (Alcohol Group Acceptor) EC 2.7.1.-
sphingosine kinase 2, human EC 2.7.1.91

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1235-1246

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier B.V. All rights reserved.

Auteurs

Andreas Weigert (A)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Andreas von Knethen (A)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Dominique Thomas (D)

Institute of Clinical Pharmacology, pharmazentrum frankfurt/ZAFES, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt am Main, Germany.

Isabel Faria (I)

Institute of Clinical Pharmacology, pharmazentrum frankfurt/ZAFES, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt am Main, Germany; Branch for Translational Medicine and Pharmacology TMP of the Fraunhofer Institute for Molecular Biology and Applied Ecology IME, 60590 Frankfurt, Germany.

Dmitry Namgaladze (D)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Ekaterina Zezina (E)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Dominik Fuhrmann (D)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Anton Petcherski (A)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Dagmar Meyer Zu Heringdorf (DMZ)

Institut für Allgemeine Pharmakologie und Toxikologie, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Heinfried H Radeke (HH)

Institut für Allgemeine Pharmakologie und Toxikologie, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany.

Bernhard Brüne (B)

Institute of Biochemistry I, Faculty of Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany; Branch for Translational Medicine and Pharmacology TMP of the Fraunhofer Institute for Molecular Biology and Applied Ecology IME, 60590 Frankfurt, Germany. Electronic address: b.bruene@biochem.uni-frankfurt.de.

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Classifications MeSH