C3a Receptor Inhibition Protects Brain Endothelial Cells Against Oxygen-glucose Deprivation/Reperfusion.
Inflammation
Ischemia
OGD
Oxidative stress
bEnd.3 cells
Journal
Experimental neurobiology
ISSN: 1226-2560
Titre abrégé: Exp Neurobiol
Pays: Korea (South)
ID NLM: 101278187
Informations de publication
Date de publication:
Apr 2019
Apr 2019
Historique:
received:
12
12
2018
revised:
09
04
2019
accepted:
10
04
2019
entrez:
30
5
2019
pubmed:
30
5
2019
medline:
30
5
2019
Statut:
ppublish
Résumé
The complement cascade is a central component of innate immunity which plays a critical role in brain inflammation. Complement C3a receptor (C3aR) is a key mediator of post-ischemic cerebral injury, and pharmacological antagonism of the C3a receptor is neuroprotective in stroke. Cerebral ischemia injures brain endothelial cells, causing blood brain barrier (BBB) disruption which further exacerbates ischemic neuronal injury. In this study, we used an
Identifiants
pubmed: 31138990
doi: 10.5607/en.2019.28.2.216
pmc: PMC6526115
doi:
Types de publication
Journal Article
Langues
eng
Pagination
216-228Déclaration de conflit d'intérêts
CONFLICT OF INTEREST: The authors report no conflict of interest with any person or Institute.
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