Impact of Weight Loss Strategies on Obesity-Induced DNA Damage.


Journal

Molecular nutrition & food research
ISSN: 1613-4133
Titre abrégé: Mol Nutr Food Res
Pays: Germany
ID NLM: 101231818

Informations de publication

Date de publication:
09 2019
Historique:
received: 11 01 2019
revised: 16 05 2019
pubmed: 30 5 2019
medline: 24 3 2020
entrez: 30 5 2019
Statut: ppublish

Résumé

Obesity causes DNA damage, which is causally related to several disorders including cancer, infertility, and cognitive dysfunctions. The aim of this study is to investigate whether weight loss improves the integrity of the genetic material. Overweight mice are fed ad libitum either with a Western diet (WD), with a 40% caloric restricted WD, or with a high carbohydrate low protein (HCLP) diet. Caloric restriction and also the HCLP diet lead to ca. 30% weight loss, which is paralleled by decreased DNA damage ("comet" formation) and oxidative damage of purines in inner organs, additionally the activity of nucleotide excision repair increased. The effects are more pronounced in animals that have received the HCLP chow. Results of biochemical analyses indicate that the reduction of DNA damage is associated with a decrease of pro-inflammatory cytokines and lower insulin levels. The study indicates that weight loss may prevent obesity-associated adverse health effects due to reduction of overall DNA damage.

Identifiants

pubmed: 31141317
doi: 10.1002/mnfr.201900045
pmc: PMC6771950
doi:

Substances chimiques

Cytokines 0
Dietary Carbohydrates 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e1900045

Subventions

Organisme : Austrian Science Fund FWF
ID : P 27129
Pays : Austria

Informations de copyright

© 2019 The Authors. Published by WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim.

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Auteurs

Tahereh Setayesh (T)

Department of Internal Medicine I, Institute of Cancer Research, Medical University of Vienna, Vienna, Austria.

Miroslav Mišík (M)

Department of Internal Medicine I, Institute of Cancer Research, Medical University of Vienna, Vienna, Austria.

Sabine A S Langie (SAS)

VITO-Health, Mol, Belgium.
Centre for Environmental Sciences, Hasselt University, Hasselt, Belgium.

Roger Godschalk (R)

Department of Pharmacology & Toxicology, School for Nutrition, Toxicology and Metabolism (NUTRIM), Maastricht University, Maastricht, The Netherlands.

Monika Waldherr (M)

Department of Internal Medicine I, Institute of Cancer Research, Medical University of Vienna, Vienna, Austria.

Thomas Bauer (T)

Department of Internal Medicine I, Institute of Cancer Research, Medical University of Vienna, Vienna, Austria.

Sabine Leitner (S)

Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria.

Christoph Bichler (C)

Section of Endocrine Surgery, Division of General Surgery, Department of Surgery, Medical University of Vienna, Vienna, Austria.

Gerhard Prager (G)

Section of Endocrine Surgery, Division of General Surgery, Department of Surgery, Medical University of Vienna, Vienna, Austria.

Georg Krupitza (G)

Clinical Institute of Pathology, Medical University of Vienna, Vienna, Austria.

Alexander Haslberger (A)

Department of Nutritional Sciences, University of Vienna, Vienna, Austria.

Siegfried Knasmüller (S)

Department of Internal Medicine I, Institute of Cancer Research, Medical University of Vienna, Vienna, Austria.

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