Combined evaluation of biomarkers as predictor of maintained remission in Crohn's disease.


Journal

World journal of gastroenterology
ISSN: 2219-2840
Titre abrégé: World J Gastroenterol
Pays: United States
ID NLM: 100883448

Informations de publication

Date de publication:
21 May 2019
Historique:
received: 07 03 2019
revised: 16 04 2019
accepted: 19 04 2019
entrez: 1 6 2019
pubmed: 1 6 2019
medline: 4 12 2019
Statut: ppublish

Résumé

The individual performances and the complementarity of Crohn's disease (CD) activity index (CDAI), C-reactive protein (CRP) and faecal calprotectin (Fcal) to monitor patients with CD remain poorly investigated in the era of "tight control" and "treat to target" strategies. To assess CDAI, CRP and Fcal variation, alone or combined, after 12 wk (W12) of anti-tumor necrosis factor (TNF) therapy to predict corticosteroids-free remission (CFREM = CDAI < 150, CRP < 2.9 mg/L and Fcal < 250 μg/g with no therapeutic intensification and no surgery) at W52. CD adult patients needing anti-TNF therapy with CDAI > 150 and either CRP > 2.9 mg/L or Fcal > 250 μg/g were prospectively enrolled. Among the 40 included patients, 13 patients (32.5%) achieved CFREM at W52. In univariable analysis, CDAI < 150 at W12 ( The combined monitoring of CDAI, CRP and Fcal after anti-TNF induction therapy is able to predict favorable outcome within one year in patients with CD.

Sections du résumé

BACKGROUND BACKGROUND
The individual performances and the complementarity of Crohn's disease (CD) activity index (CDAI), C-reactive protein (CRP) and faecal calprotectin (Fcal) to monitor patients with CD remain poorly investigated in the era of "tight control" and "treat to target" strategies.
AIM OBJECTIVE
To assess CDAI, CRP and Fcal variation, alone or combined, after 12 wk (W12) of anti-tumor necrosis factor (TNF) therapy to predict corticosteroids-free remission (CFREM = CDAI < 150, CRP < 2.9 mg/L and Fcal < 250 μg/g with no therapeutic intensification and no surgery) at W52.
METHODS METHODS
CD adult patients needing anti-TNF therapy with CDAI > 150 and either CRP > 2.9 mg/L or Fcal > 250 μg/g were prospectively enrolled.
RESULTS RESULTS
Among the 40 included patients, 13 patients (32.5%) achieved CFREM at W52. In univariable analysis, CDAI < 150 at W12 (
CONCLUSION CONCLUSIONS
The combined monitoring of CDAI, CRP and Fcal after anti-TNF induction therapy is able to predict favorable outcome within one year in patients with CD.

Identifiants

pubmed: 31148906
doi: 10.3748/wjg.v25.i19.2354
pmc: PMC6529885
doi:

Substances chimiques

Biomarkers 0
Gastrointestinal Agents 0
Leukocyte L1 Antigen Complex 0
TNF protein, human 0
Tumor Necrosis Factor-alpha 0
C-Reactive Protein 9007-41-4

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

2354-2364

Déclaration de conflit d'intérêts

Conflict-of-interest statement: Buisson A declares lecture fees for MSD, Abbvie, Ferring, Takeda, Vifor Pharma, Hospira and consulting fees for Abbvie, Takeda and Hospira. Bouguen G received lecture fees from Abbvie, Ferring, MSD, Takeda and Pfizer and consulting fees from Takeda and Janssen. The other authors declare no conflict of interest related to this work.

Références

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Auteurs

Elisa Sollelis (E)

Inserm 3iHP, CHU Clermont-Ferrand, Service d'Hépato-Gastro Entérologie, Université Clermont Auvergne, Clermont-Ferrand F-63000, France.

Régine Minet Quinard (RM)

Biochemistry laboratory, University Hospital G. Montpied, Clermont-Ferrand F-63000, France.

Guillaume Bouguen (G)

CHU Rennes, Univ Rennes, INSERM, CIC1414, Institut NUMECAN (Nutrition Metabolisms and Cancer), Rennes F-35000, France.

Marion Goutte (M)

Inserm 3iHP, CHU Clermont-Ferrand, Service d'Hépato-Gastro Entérologie, Université Clermont Auvergne, Clermont-Ferrand F-63000, France.

Félix Goutorbe (F)

Gastroenterology Department, Hospital of Bayonne, Bayonne F-64100, France.

Damien Bouvier (D)

Biochemistry laboratory, University Hospital G. Montpied, Clermont-Ferrand F-63000, France.

Bruno Pereira (B)

Biostatistics Unit, DRCI, University Hospital, Clermont-Ferrand F-63000, France.

Gilles Bommelaer (G)

Inserm 3iHP, CHU Clermont-Ferrand, Service d'Hépato-Gastro Entérologie, Université Clermont Auvergne, Clermont-Ferrand F-63000, France.

Anthony Buisson (A)

Inserm 3iHP, CHU Clermont-Ferrand, Service d'Hépato-Gastro Entérologie, Université Clermont Auvergne, Clermont-Ferrand F-63000, France. a_buisson@hotmail.fr.

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Classifications MeSH