Long-term metformin use may improve clinical outcomes in diabetic patients with non-alcoholic steatohepatitis and bridging fibrosis or compensated cirrhosis.


Journal

Alimentary pharmacology & therapeutics
ISSN: 1365-2036
Titre abrégé: Aliment Pharmacol Ther
Pays: England
ID NLM: 8707234

Informations de publication

Date de publication:
08 2019
Historique:
received: 07 02 2019
revised: 18 03 2019
accepted: 10 05 2019
pubmed: 4 6 2019
medline: 15 5 2020
entrez: 4 6 2019
Statut: ppublish

Résumé

Metformin may protect against hepatocellular carcinoma and mortality among patients with type 2 diabetes. To investigate whether long-term use of metformin improves survival and reduces liver-related outcomes among patients with type 2 diabetes and non-alcoholic steatohepatitis. A total of 191 diabetic patients with biopsy-proven non-alcoholic steatohepatitis and bridging fibrosis or compensated cirrhosis were retrospectively identified at Indiana University Medical Center between October 2004 and January 2016. Of them, 110 were users and 81 never-users of metformin. Primary outcomes were transplant-free survival, development of hepatocellular carcinoma or a first event of hepatic decompensation. Cirrhosis was present in 85% of metformin users and 88% of nonusers. Metformin dose was greater than or equal to 1 g/d in 104 out of 110 users and its median duration of use was 6 (95% CI: 4.4-7.9) years. The mean follow-up was 6.92 and 6.80 years for metformin users and non-users, respectively. During follow-up, 28 patients developed hepatocellular carcinoma (metformin users: 7, nonusers: 21), and 52 died (metformin users: 7, nonusers: 24) or were transplanted (metformin users: 13, non-users: 13). Metformin use was associated with lower risk of overall mortality or transplant (HR: 0.42; 95% CI: 0.24-0.74, P = 0.003) and hepatocellular carcinoma (sHR: 0.25; 95% CI: 0.11-0.58, P = 0.001), and remained independently associated with both outcomes after propensity-score and covariate-adjusted analyses. No instances of hepatotoxicity or lactic acidosis were observed. Our study demonstrated an association between long-term metformin use and reduced the risk of all-cause mortality/transplant and hepatocellular carcinoma in diabetics with non-alcoholic steatohepatitis and advanced fibrosis.

Sections du résumé

BACKGROUND
Metformin may protect against hepatocellular carcinoma and mortality among patients with type 2 diabetes.
AIM
To investigate whether long-term use of metformin improves survival and reduces liver-related outcomes among patients with type 2 diabetes and non-alcoholic steatohepatitis.
METHODS
A total of 191 diabetic patients with biopsy-proven non-alcoholic steatohepatitis and bridging fibrosis or compensated cirrhosis were retrospectively identified at Indiana University Medical Center between October 2004 and January 2016. Of them, 110 were users and 81 never-users of metformin. Primary outcomes were transplant-free survival, development of hepatocellular carcinoma or a first event of hepatic decompensation.
RESULTS
Cirrhosis was present in 85% of metformin users and 88% of nonusers. Metformin dose was greater than or equal to 1 g/d in 104 out of 110 users and its median duration of use was 6 (95% CI: 4.4-7.9) years. The mean follow-up was 6.92 and 6.80 years for metformin users and non-users, respectively. During follow-up, 28 patients developed hepatocellular carcinoma (metformin users: 7, nonusers: 21), and 52 died (metformin users: 7, nonusers: 24) or were transplanted (metformin users: 13, non-users: 13). Metformin use was associated with lower risk of overall mortality or transplant (HR: 0.42; 95% CI: 0.24-0.74, P = 0.003) and hepatocellular carcinoma (sHR: 0.25; 95% CI: 0.11-0.58, P = 0.001), and remained independently associated with both outcomes after propensity-score and covariate-adjusted analyses. No instances of hepatotoxicity or lactic acidosis were observed.
CONCLUSION
Our study demonstrated an association between long-term metformin use and reduced the risk of all-cause mortality/transplant and hepatocellular carcinoma in diabetics with non-alcoholic steatohepatitis and advanced fibrosis.

Identifiants

pubmed: 31157422
doi: 10.1111/apt.15331
doi:

Substances chimiques

Metformin 9100L32L2N

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

317-328

Commentaires et corrections

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Informations de copyright

© 2019 John Wiley & Sons Ltd.

Auteurs

Eduardo Vilar-Gomez (E)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

Raj Vuppalanchi (R)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

Archita P Desai (AP)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

Samer Gawrieh (S)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

Marwan Ghabril (M)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

Romil Saxena (R)

Department of Pathology, Indiana University School of Medicine, Indianapolis, Indiana.

Oscar W Cummings (OW)

Department of Pathology, Indiana University School of Medicine, Indianapolis, Indiana.

Naga Chalasani (N)

Division of Gastroenterology and Hepatology, Indiana University School of Medicine, Indianapolis, Indiana.

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