Chemokine Signaling in Chemotherapy-Induced Neuropathic Pain.
central nervous system
chemokines
chemotherapy
cytokines
inflammatory mediators
peripheral nervous system
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
14 Jun 2019
14 Jun 2019
Historique:
received:
10
05
2019
revised:
10
06
2019
accepted:
12
06
2019
entrez:
15
6
2019
pubmed:
15
6
2019
medline:
28
11
2019
Statut:
epublish
Résumé
Chemotherapy-induced peripheral neuropathy (CIPN) is a side effect of chemotherapics such as taxanes, vinca alkaloids, and platinum compounds. In recent years, several reports have indicated the involvement of different molecular mechanisms in CIPN. The pathways described so far are diverse and target various components of the peripheral Nervous System (PNS). Among the contributors to neuropathic pain, inflammation has been indicated as a powerful driver of CIPN. Several pieces of evidence have demonstrated a chemotherapy-induced increase in peripheral pro-inflammatory cytokines and a strong correlation with peripheral neuropathy. At present, there are not adequate strategies to prevent CIPN, although there are drugs for treating CIPN, such as duloxetine, that have displayed a moderate effect on CIPN. In this review, we focus on the players involved in CIPN with a particular emphasis on chemokine signaling.
Identifiants
pubmed: 31197114
pii: ijms20122904
doi: 10.3390/ijms20122904
pmc: PMC6627296
pii:
doi:
Substances chimiques
Antineoplastic Agents
0
Chemokines
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
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