Superoxide and NADPH oxidase do not modulate skin blood flow in older exercising adults with and without type 2 diabetes.


Journal

Microvascular research
ISSN: 1095-9319
Titre abrégé: Microvasc Res
Pays: United States
ID NLM: 0165035

Informations de publication

Date de publication:
09 2019
Historique:
received: 09 04 2019
revised: 09 05 2019
accepted: 11 06 2019
pubmed: 15 6 2019
medline: 6 5 2020
entrez: 15 6 2019
Statut: ppublish

Résumé

High aerobic fitness may prevent age-related decrements in cutaneous vasodilation while type 2 diabetes may exacerbate this decline. The mechanisms underlying these responses remain unclear, but may be due to an excess of reactive oxygen species. We hypothesized that superoxide scavenging or NADPH oxidase inhibition would improve cutaneous vasodilation in older adults exercising in the heat, particularly in healthy low-fit individuals and those with type 2 diabetes. Twenty seven older adults were evenly separated into three groups (healthy low-fit: VO In all groups, L-NAME consistently reduced CVC Superoxide and NADPH oxidase do not modulate cutaneous vasodilation in healthy low- or high-fit older adults exercising in the heat, regardless of aerobic fitness level or relative exercise intensity employed, nor do they influence cutaneous vasodilation during an exercise-heat stress in those with type 2 diabetes. However, NOS remains an important modulator of cutaneous vasodilation during exercise in all groups.

Identifiants

pubmed: 31199960
pii: S0026-2862(19)30080-9
doi: 10.1016/j.mvr.2019.103886
pii:
doi:

Substances chimiques

Enzyme Inhibitors 0
Free Radical Scavengers 0
Superoxides 11062-77-4
Nitric Oxide 31C4KY9ESH
Nitric Oxide Synthase EC 1.14.13.39
NADPH Oxidases EC 1.6.3.-

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

103886

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Gregory W McGarr (GW)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada.

Naoto Fujii (N)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada; Faculty of Health and Sport Sciences, University of Tsukuba, Tsukuba City, Japan.

Brendan D McNeely (BD)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada.

Kion Hatam (K)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada.

Takeshi Nishiyasu (T)

Faculty of Health and Sport Sciences, University of Tsukuba, Tsukuba City, Japan.

Ronald J Sigal (RJ)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada; Departments of Medicine, Cardiac Sciences and Community Health Sciences, Faculties of Medicine and Kinesiology, University of Calgary, Calgary, Alberta, Canada; Clinical Epidemiology Program, Ottawa Hospital Research Institute, Ottawa, Ontario, Canada.

Pierre Boulay (P)

Faculty of Physical Activity Sciences, University of Sherbrooke, Sherbrooke, Quebec, Canada.

Glen P Kenny (GP)

Human and Environmental Physiology Research Unit, School of Human Kinetics, University of Ottawa, Ottawa, Ontario, Canada; Clinical Epidemiology Program, Ottawa Hospital Research Institute, Ottawa, Ontario, Canada. Electronic address: gkenny@uottawa.ca.

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