Proliferative memory SAMHD1low CD4+ T cells harbour high levels of HIV-1 with compartmentalized viral populations.


Journal

PLoS pathogens
ISSN: 1553-7374
Titre abrégé: PLoS Pathog
Pays: United States
ID NLM: 101238921

Informations de publication

Date de publication:
06 2019
Historique:
received: 05 12 2018
accepted: 24 05 2019
revised: 02 07 2019
pubmed: 21 6 2019
medline: 4 12 2019
entrez: 21 6 2019
Statut: epublish

Résumé

We previously reported the presence of memory CD4+ T cells that express low levels of SAMHD1 (SAMHD1low) in peripheral blood and lymph nodes from both HIV-1 infected and uninfected individuals. These cells are enriched in Th17 and Tfh subsets, two populations known to be preferentially targeted by HIV-1. Here we investigated whether SAMHD1low CD4+ T-cells harbour replication-competent virus and compartimentalized HIV-1 genomes. We sorted memory CD4+CD45RO+SAMHD1low, CD4+CD45RO+SAMHD1+ and naive CD4+CD45RO-SAMHD1+ cells from HIV-1-infected patients on anti-retroviral therapy (c-ART) and performed HIV-1 DNA quantification, ultra-deep-sequencing of partial env (C2/V3) sequences and phenotypic characterization of the cells. We show that SAMHD1low cells include novel Th17 CCR6+ subsets that lack CXCR3 and CCR4 (CCR6+DN). There is a decrease of the % of Th17 in SAMHD1low compartment in infected compared to uninfected individuals (41% vs 55%, p<0.05), whereas the % of CCR6+DN increases (7.95% vs 3.8%, p<0.05). Moreover, in HIV-1 infected patients, memory SAMHD1low cells harbour high levels of HIV-1 DNA compared to memory SAMHD1+ cells (4.5 vs 3.8 log/106cells, respectively, p<0.001), while naïve SAMHD1+ showed significantly lower levels (3.1 log/106cells, p<0.0001). Importantly, we show that SAMHD1low cells contain p24-producing cells. Moreover, phylogenetic analyses revealed well-segregated HIV-1 DNA populations with compartmentalization between SAMHD1low and SAMHD1+ memory cells, and limited viral exchange. As expected, the % of Ki67+ cells was significantly higher in SAMHD1low compared to SAMHD1+ cells. There was positive association between levels of HIV-1 DNA and Ki67+ in memory SAMHD1low cells, but not in memory and naïve SAMHD1+ CD4+ T-cells. Altogether, these data suggest that proliferative memory SAMHD1low cells contribute to viral persistence.

Identifiants

pubmed: 31220191
doi: 10.1371/journal.ppat.1007868
pii: PPATHOGENS-D-18-02327
pmc: PMC6605680
doi:

Substances chimiques

Anti-Retroviral Agents 0
SAM Domain and HD Domain-Containing Protein 1 EC 3.1.5.-
SAMHD1 protein, human EC 3.1.5.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e1007868

Subventions

Organisme : NIAID NIH HHS
ID : R21 AI131971
Pays : United States

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Lylia Hani (L)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.

Antoine Chaillon (A)

Vaccine Research Institute (VRI), Créteil, France.
Department of Medicine, University of California San Diego, CA, United States of America.

Marie-Laure Nere (ML)

Hôpital Saint Louis, INSERM U944, Université de Paris, Paris, France.

Nicolas Ruffin (N)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.

Joudy Alameddine (J)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.

Maud Salmona (M)

Hôpital Saint Louis, INSERM U944, Université de Paris, Paris, France.

José-Luiz Lopez Zaragoza (JL)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.
AP-HP, Hôpital H. Mondor-A. Chenevier, Service d'immunologie clinique et maladies infectieuses, Créteil, France.

Davey M Smith (DM)

Department of Medicine, University of California San Diego, CA, United States of America.

Olivier Schwartz (O)

Vaccine Research Institute (VRI), Créteil, France.
Unité Virus et Immunité, Département de Virologie, Institut Pasteur, Paris, France.

Jean-Daniel Lelièvre (JD)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.
AP-HP, Hôpital H. Mondor-A. Chenevier, Service d'immunologie clinique et maladies infectieuses, Créteil, France.

Constance Delaugerre (C)

Vaccine Research Institute (VRI), Créteil, France.
Hôpital Saint Louis, INSERM U944, Université de Paris, Paris, France.

Yves Lévy (Y)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.
AP-HP, Hôpital H. Mondor-A. Chenevier, Service d'immunologie clinique et maladies infectieuses, Créteil, France.

Nabila Seddiki (N)

Inserm, U955 Equipe 16, Créteil, France.
Université Paris Est, Faculté de médecine, Créteil, France.
Vaccine Research Institute (VRI), Créteil, France.

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