The l-isoaspartate modification within protein fragments in the aging lens can promote protein aggregation.


Journal

The Journal of biological chemistry
ISSN: 1083-351X
Titre abrégé: J Biol Chem
Pays: United States
ID NLM: 2985121R

Informations de publication

Date de publication:
09 08 2019
Historique:
received: 25 04 2019
revised: 05 06 2019
pubmed: 27 6 2019
medline: 17 3 2020
entrez: 27 6 2019
Statut: ppublish

Résumé

Transparency in the lens is accomplished by the dense packing and short-range order interactions of the crystallin proteins in fiber cells lacking organelles. These features are accompanied by a lack of protein turnover, leaving lens proteins susceptible to a number of damaging modifications and aggregation. The loss of lens transparency is attributed in part to such aggregation during aging. Among the damaging post-translational modifications that accumulate in long-lived proteins, isomerization at aspartate residues has been shown to be extensive throughout the crystallins. In this study of the human lens, we localize the accumulation of l-isoaspartate within water-soluble protein extracts primarily to crystallin peptides in high-molecular weight aggregates and show with MS that these peptides are from a variety of crystallins. To investigate the consequences of aspartate isomerization, we investigated two αA crystallin peptides

Identifiants

pubmed: 31239355
pii: S0021-9258(20)30169-1
doi: 10.1074/jbc.RA119.009052
pmc: PMC6690693
pii:
doi:

Substances chimiques

Crystallins 0
Isoaspartic Acid 0
Peptides 0
Protein Aggregates 0
Recombinant Proteins 0
alpha-Crystallin A Chain 0
alpha-Crystallin B Chain 0
PCMT1 protein, human EC 2.1.1.77
Protein D-Aspartate-L-Isoaspartate Methyltransferase EC 2.1.1.77

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

12203-12219

Subventions

Organisme : NEI NIH HHS
ID : R01 EY008313
Pays : United States
Organisme : NEI NIH HHS
ID : R01 EY023588
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM007185
Pays : United States

Informations de copyright

© 2019 Warmack et al.

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Auteurs

Rebeccah A Warmack (RA)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095.

Harrison Shawa (H)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095.

Kate Liu (K)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095.

Katia Lopez (K)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095.

Joseph A Loo (JA)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095.

Joseph Horwitz (J)

Molecular Biology Institute, UCLA, Los Angeles, California 90095; Jules Stein Eye Institute, UCLA, Los Angeles, California 90095.

Steven G Clarke (SG)

Department of Chemistry and Biochemistry, UCLA, Los Angeles, California 90095; Molecular Biology Institute, UCLA, Los Angeles, California 90095. Electronic address: Clarke@mbi.ucla.edu.

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Classifications MeSH