STAT5a/b Deficiency Delays, but does not Prevent, Prolactin-Driven Prostate Tumorigenesis in Mice.

AKT ERK1/2 STAT5 androgens cell hierarchy escape mechanisms knockout prolactin prostate cancer stem/progenitor cells

Journal

Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829

Informations de publication

Date de publication:
02 Jul 2019
Historique:
received: 11 06 2019
revised: 28 06 2019
accepted: 28 06 2019
entrez: 5 7 2019
pubmed: 5 7 2019
medline: 5 7 2019
Statut: epublish

Résumé

The canonical prolactin (PRL) Signal Transducer and Activator of Transcription (STAT) 5 pathway has been suggested to contribute to human prostate tumorigenesis via an autocrine/paracrine mechanism. The probasin (Pb)-PRL transgenic mouse models this mechanism by overexpressing PRL specifically in the prostate epithelium leading to strong STAT5 activation in luminal cells. These mice exhibit hypertrophic prostates harboring various pre-neoplastic lesions that aggravate with age and accumulation of castration-resistant stem/progenitor cells. As STAT5 signaling is largely predominant over other classical PRL-triggered pathways in Pb-PRL prostates, we reasoned that Pb-Cre recombinase-driven genetic deletion of a floxed

Identifiants

pubmed: 31269779
pii: cancers11070929
doi: 10.3390/cancers11070929
pmc: PMC6678910
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : Institut National Du Cancer
ID : INCa_6672
Organisme : Institut National de la Santé et de la Recherche Médicale
ID : Recurrent funding
Organisme : Université Paris Descartes
ID : Recurrent funding
Organisme : Centre National de la Recherche Scientifique
ID : Recurrent funding
Organisme : Austrian Science Fund
ID : SFB-F4707 and SFB-F06105

Déclaration de conflit d'intérêts

The authors declare no conflict of interest.

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Auteurs

Florence Boutillon (F)

Institut Necker Enfants Malades, Inserm U1151, 75014 Paris, France.
Faculté de médecine, Université Paris Descartes, 75014 Paris, France.

Natascha Pigat (N)

Institut Necker Enfants Malades, Inserm U1151, 75014 Paris, France.
Faculté de médecine, Université Paris Descartes, 75014 Paris, France.

Lucila Sackmann Sala (LS)

Institut Necker Enfants Malades, Inserm U1151, 75014 Paris, France.
Faculté de médecine, Université Paris Descartes, 75014 Paris, France.

Edouard Reyes-Gomez (E)

Unité d'Histologie et d'Anatomie Pathologique, Ecole Nationale Vétérinaire d'Alfort, 94704 Maisons-Alfort, France.
Laboratoire d'anatomo-cytopathologie, BioPôle Alfort, Ecole Nationale Vétérinaire d'Alfort, 94704 Maisons-Alfort, France.
U955-IMRB, Inserm, Ecole Nationale Vétérinaire d'Alfort, UPEC, 94704 Maisons-Alfort, France.

Richard Moriggl (R)

Institute of Animal Breeding and Genetics, University of Veterinary Medicine Vienna, 1210 Vienna, Austria.
Medical University of Vienna, 1090 Vienna, Austria.

Jacques-Emmanuel Guidotti (JE)

Institut Necker Enfants Malades, Inserm U1151, 75014 Paris, France.
Faculté de médecine, Université Paris Descartes, 75014 Paris, France.

Vincent Goffin (V)

Institut Necker Enfants Malades, Inserm U1151, 75014 Paris, France. Vincent.goffin@inserm.fr.
Faculté de médecine, Université Paris Descartes, 75014 Paris, France. Vincent.goffin@inserm.fr.

Classifications MeSH