Mibefradil, a T-type Ca


Journal

British journal of pharmacology
ISSN: 1476-5381
Titre abrégé: Br J Pharmacol
Pays: England
ID NLM: 7502536

Informations de publication

Date de publication:
10 2019
Historique:
received: 29 10 2018
revised: 21 06 2019
accepted: 28 06 2019
pubmed: 5 7 2019
medline: 22 9 2020
entrez: 5 7 2019
Statut: ppublish

Résumé

Mibefradil, a T-type Ca Human Orai1-3 cDNAs in tetracycline-regulated pcDNA4/TO vectors were transfected into HEK293 T-REx cells with stromal interaction molecule 1 (STIM1) stable expression. The Orai currents were recorded by whole-cell and excised-membrane patch clamp. Ca Mibefradil inhibited Orai1, Orai2, and Orai3 currents dose-dependently. The IC Mibefradil is a potent cell surface blocker of Orai channels, demonstrating a new pharmacological action of this compound in regulating cell growth and death, which could be relevant to its anti-cancer activity.

Sections du résumé

BACKGROUND AND PURPOSE
Mibefradil, a T-type Ca
EXPERIMENTAL APPROACH
Human Orai1-3 cDNAs in tetracycline-regulated pcDNA4/TO vectors were transfected into HEK293 T-REx cells with stromal interaction molecule 1 (STIM1) stable expression. The Orai currents were recorded by whole-cell and excised-membrane patch clamp. Ca
KEY RESULTS
Mibefradil inhibited Orai1, Orai2, and Orai3 currents dose-dependently. The IC
CONCLUSIONS AND IMPLICATIONS
Mibefradil is a potent cell surface blocker of Orai channels, demonstrating a new pharmacological action of this compound in regulating cell growth and death, which could be relevant to its anti-cancer activity.

Identifiants

pubmed: 31271653
doi: 10.1111/bph.14788
pmc: PMC6780027
doi:

Substances chimiques

Calcium Channel Blockers 0
Calcium Channels 0
ORAI1 Protein 0
ORAI1 protein, human 0
ORAI2 Protein 0
ORAI2 protein, human 0
Orai3 protein, human 0
Mibefradil 27B90X776A
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3845-3856

Informations de copyright

© 2019 The British Pharmacological Society.

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Auteurs

Pengyun Li (P)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.
Key Laboratory of Medical Electrophysiology, Ministry of Education, and Institute of Cardiovascular Research, Southwest Medical University, Luzhou, China.

Hussein N Rubaiy (HN)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.

Gui-Lan Chen (GL)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.
Key Laboratory of Medical Electrophysiology, Ministry of Education, and Institute of Cardiovascular Research, Southwest Medical University, Luzhou, China.

Thomas Hallett (T)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.

Nawel Zaibi (N)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.

Bo Zeng (B)

Key Laboratory of Medical Electrophysiology, Ministry of Education, and Institute of Cardiovascular Research, Southwest Medical University, Luzhou, China.

Rahul Saurabh (R)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.

Shang-Zhong Xu (SZ)

Centre for Atherothrombosis and Metabolic Disease, Hull York Medical School, University of Hull, Hull, UK.

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Classifications MeSH