Polymorphisms in MMP-14 and MMP-2 genes and ovarian cancer survival.


Journal

Cancer biomarkers : section A of Disease markers
ISSN: 1875-8592
Titre abrégé: Cancer Biomark
Pays: Netherlands
ID NLM: 101256509

Informations de publication

Date de publication:
2019
Historique:
pubmed: 10 7 2019
medline: 7 1 2020
entrez: 9 7 2019
Statut: ppublish

Résumé

Functional polymorphisms in matrix metalloproteinases can increase or decrease the risk of cancer. This study focused on ovarian cancer and investigated how polymorphisms in the coding region of MMP-14 and the promoter region of MMP-2 are related to clinical characteristics including survival. In 144 patients with ovarian tumours from a Caucasian population, polymorphisms of MMP-14 (+7096 and +6767) and MMP-2 (-735 and -1306) were analysed. These results were then correlated to the immunohistochemical expression of MMP-14 and MMP-2 and clinical characteristics. In these patients, the MMP-14 +7096 polymorphism showed only TT genotype, in sharp contrast to the described MAF (minimal allele frequency) C of 27%. The MMP-14 +6767 G>A polymorphism was found to have a hazard ratio of 2.09 (CI 1.00-4.35, p 0.046) for recurrence-free survival in advanced-stage patients. However, this significance disappeared after Bonferroni correction for multiple testing. No other correlations between MMP-14 and MMP-2 polymorphisms, immunohistochemistry and clinical characteristics were found, except between the MMP-2 -1306 polymorphism and differentiation grade, with a Spearman correlation coefficient of -0.19, p 0.064. In ovarian cancer, the MMP-14 +6767 G>A polymorphism in the coding region seems to improve recurrence-free survival with a hazard ratio of 2.09 (CI 1.00-4.35, p 0.046). However, as this significance disappeared after correction for multiple testing, there is a need for further research on the functional effect of this change in the MMP-14 gene with larger patient sample sizes.

Sections du résumé

BACKGROUND BACKGROUND
Functional polymorphisms in matrix metalloproteinases can increase or decrease the risk of cancer. This study focused on ovarian cancer and investigated how polymorphisms in the coding region of MMP-14 and the promoter region of MMP-2 are related to clinical characteristics including survival.
METHODS METHODS
In 144 patients with ovarian tumours from a Caucasian population, polymorphisms of MMP-14 (+7096 and +6767) and MMP-2 (-735 and -1306) were analysed. These results were then correlated to the immunohistochemical expression of MMP-14 and MMP-2 and clinical characteristics.
RESULTS RESULTS
In these patients, the MMP-14 +7096 polymorphism showed only TT genotype, in sharp contrast to the described MAF (minimal allele frequency) C of 27%. The MMP-14 +6767 G>A polymorphism was found to have a hazard ratio of 2.09 (CI 1.00-4.35, p 0.046) for recurrence-free survival in advanced-stage patients. However, this significance disappeared after Bonferroni correction for multiple testing. No other correlations between MMP-14 and MMP-2 polymorphisms, immunohistochemistry and clinical characteristics were found, except between the MMP-2 -1306 polymorphism and differentiation grade, with a Spearman correlation coefficient of -0.19, p 0.064.
CONCLUSIONS CONCLUSIONS
In ovarian cancer, the MMP-14 +6767 G>A polymorphism in the coding region seems to improve recurrence-free survival with a hazard ratio of 2.09 (CI 1.00-4.35, p 0.046). However, as this significance disappeared after correction for multiple testing, there is a need for further research on the functional effect of this change in the MMP-14 gene with larger patient sample sizes.

Identifiants

pubmed: 31282404
pii: CBM181826
doi: 10.3233/CBM-181826
doi:

Substances chimiques

Matrix Metalloproteinase 2 EC 3.4.24.24
Matrix Metalloproteinase 14 EC 3.4.24.80

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

233-241

Auteurs

M Caroline Vos (MC)

Department of Obstetrics and Gynaecology, Elisabeth-Tweesteden Hospital, 5000 LC Tilburg, the Netherlands.

Angela van Tilborg (A)

Department of Obstetrics and Gynaecology and Department of Pathology, Radboud University Nijmegen Medical Centre, 6500 HB Nijmegen, the Netherlands.

William J Brands (WJ)

Department of Pathology, Elisabeth-Tweesteden Hospital, 5000 LC Tilburg, the Netherlands.

Dorry Boll (D)

Department of Obstetrics and Gynaecology, Elisabeth-Tweesteden Hospital, 5000 LC Tilburg, the Netherlands.

Dennis van Hamont (D)

Department of Obstetrics and Gynaecology, Amphia Ziekenhuis, Breda, the Netherlands.

Hans van der Putten (H)

Department of Obstetrics and Gynaecology, Catharina Hospital, Eindhoven, the Netherlands.

Brenda Pijlman (B)

Department of Obstetrics and Gynaecology, Jeroen Bosch Ziekenhuis, s-Hertogenbosch, the Netherlands.

Anneke A M van der Wurff (AAM)

Department of Pathology, Elisabeth-Tweesteden Hospital, 5000 LC Tilburg, the Netherlands.

Toin H van Kuppevelt (TH)

Department of Biochemistry, Radboud Institute for Molecular Life Sciences, Radboud University Medical Centre, 6500 HB Nijmegen, the Netherlands.

Leon F A G Massuger (LFAG)

Department of Obstetrics and Gynaecology, Radboud University Nijmegen Medical Centre, 6500 HB Nijmegen, the Netherlands.

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Classifications MeSH