Electroacupuncture Pretreatment Ameliorates PTSD-Like Behaviors in Rats by Enhancing Hippocampal Neurogenesis via the Keap1/Nrf2 Antioxidant Signaling Pathway.

electroacupuncture hippocampus keap1/Nrf2 post-traumatic stress disorder pretreatment

Journal

Frontiers in cellular neuroscience
ISSN: 1662-5102
Titre abrégé: Front Cell Neurosci
Pays: Switzerland
ID NLM: 101477935

Informations de publication

Date de publication:
2019
Historique:
received: 17 01 2019
accepted: 06 06 2019
entrez: 12 7 2019
pubmed: 12 7 2019
medline: 12 7 2019
Statut: epublish

Résumé

Electroacupuncture (EA) pretreatment is a clinically useful therapy for several brain disorders. However, whether and via which exact molecular mechanisms it ameliorates post-traumatic stress disorder (PTSD) remains unclear. In the present study, rats received EA stimulation for seven consecutive days before exposure to enhanced single prolonged stress (ESPS). Anxiety-like and fear learning behaviors; hippocampal neurogenesis; the expression of nuclear factor erythroid 2-related factor 2 (Nrf2), Kelch-like ECH-associated protein 1 (keap1), and heme oxygenase 1 (HO-1); and the activity of AMP-activated kinase (AMPK) were evaluated at 14 days after ESPS. EA pretreatment improved hippocampal neurogenesis and ameliorated anxiety-like behaviors in ESPS-treated rats. EA pretreatment also increased the expression of Nrf2 and HO-1 and the activity of AMPK. Furthermore, Nrf2 knockdown by a short hairpin RNA affected anxiety-like behaviors and expression of neuroprotective markers (BDNF, DCX) in a manner similar to ESPS alone and dampened the neuroprotective effects of EA pretreatment. In contrast, Keap1 knockdown increased the expression of HO-1, improved hippocampal neurogenesis, and alleviated PTSD-like behaviors. Altogether, our results suggest that EA pretreatment ameliorates ESPS-induced anxiety-like behaviors and prevents hippocampal neurogenesis disruption in a rat model of PTSD possibly through regulation of the keap1/Nrf2 antioxidant defense pathway.

Identifiants

pubmed: 31293390
doi: 10.3389/fncel.2019.00275
pmc: PMC6598452
doi:

Types de publication

Journal Article

Langues

eng

Pagination

275

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Auteurs

Cui-Hong Zhou (CH)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Fen Xue (F)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Shan-Shan Xue (SS)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Han-Fei Sang (HF)

Department of Anesthesiology, Xiang'an Hospital, Xiamen, China.

Ling Liu (L)

Institution of Neuroscience, Fourth Military Medical University, Xi'an, China.

Ying Wang (Y)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Min Cai (M)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Zhang-Jin Zhang (ZJ)

School of Chinese Medicine, LKS Faculty of Medicine, The University of Hong Kong, Hong Kong, China.

Qing-Rong Tan (QR)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Hua-Ning Wang (HN)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Zheng-Wu Peng (ZW)

Xijing Hospital, Fourth Military Medical University, Xi'an, China.

Classifications MeSH