Store-operated calcium entry (SOCE) contributes to phosphorylation of p38 MAPK and suppression of TNF-α signalling in the intestinal epithelial cells.
Muscarinic acetylcholine receptor
Protease-activated receptor-2
Store-operated calcium entry
Tumor necrosis factor
p38 MAPK
Journal
Cellular signalling
ISSN: 1873-3913
Titre abrégé: Cell Signal
Pays: England
ID NLM: 8904683
Informations de publication
Date de publication:
11 2019
11 2019
Historique:
received:
15
03
2019
revised:
25
06
2019
accepted:
06
07
2019
pubmed:
12
7
2019
medline:
9
9
2020
entrez:
12
7
2019
Statut:
ppublish
Résumé
Calcium influx via store-operated calcium entry (SOCE) has an important role for regulation of vast majority of cellular physiological events. MAPK signalling is also another pivotal modulator of many cellular functions. However, the relationship between SOCE and MAPK is not well understood. In this study, we elucidated the involvement of SOCE in Gα
Identifiants
pubmed: 31295519
pii: S0898-6568(19)30154-8
doi: 10.1016/j.cellsig.2019.109358
pii:
doi:
Substances chimiques
NF-kappa B
0
Receptor, Muscarinic M3
0
Receptor, PAR-2
0
Receptors, Histamine H1
0
Tumor Necrosis Factor-alpha
0
p38 Mitogen-Activated Protein Kinases
EC 2.7.11.24
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
109358Informations de copyright
Copyright © 2019 Elsevier Inc. All rights reserved.