Degranulation of human cytotoxic lymphocytes is a major source of proteolytically active soluble CD26/DPP4.


Journal

Cellular and molecular life sciences : CMLS
ISSN: 1420-9071
Titre abrégé: Cell Mol Life Sci
Pays: Switzerland
ID NLM: 9705402

Informations de publication

Date de publication:
Feb 2020
Historique:
received: 12 02 2019
accepted: 24 06 2019
revised: 14 06 2019
pubmed: 14 7 2019
medline: 7 3 2020
entrez: 14 7 2019
Statut: ppublish

Résumé

Dipeptidyl peptidase 4 (DPP4, CD26) is a serine protease detected on several immune cells and on epithelial cells of various organs. Besides the membrane-bound enzyme, a catalytically active soluble form (sCD26/DPP4) is detected in several body fluids. Both variants cleave off dipeptides from the N-termini of various chemokines, neuropeptides, and hormones. CD26/DPP4 plays a fundamental role in the regulation of blood glucose levels by inactivating insulinotropic incretins and CD26/DPP4 inhibitors are thus routinely used in diabetes mellitus type 2 therapy to improve glucose tolerance. Such inhibitors might also prevent the CD26/DPP4-mediated inactivation of the T-cell chemoattractant CXCL10 released by certain tumors and thus improve anti-tumor immunity and immunotherapy. Despite its implication in the regulation of many (patho-)physiological processes and its consideration as a biomarker and therapeutic target, the cellular source of sCD26/DPP4 remains highly debated and mechanisms of its release are so far unknown. In line with recent reports that activated T lymphocytes could be a major source of sCD26/DPP4, we now demonstrate that CD26/DPP4 is stored in secretory granules of several major human cytotoxic lymphocyte populations and co-localizes with effector proteins such as granzymes, perforin, and granulysin. Upon stimulation, vesicular CD26/DPP4 is rapidly translocated to the cell surface in a Ca

Identifiants

pubmed: 31300870
doi: 10.1007/s00018-019-03207-0
pii: 10.1007/s00018-019-03207-0
doi:

Substances chimiques

DPP4 protein, human EC 3.4.14.5
Dipeptidyl Peptidase 4 EC 3.4.14.5
Calcium SY7Q814VUP

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

751-764

Subventions

Organisme : Deutsche Forschungsgemeinschaft
ID : JA 610 7/1
Organisme : Deutsche Forschungsgemeinschaft
ID : Ka 502/19-1
Organisme : Deutsche Forschungsgemeinschaft
ID : Cluster of Excellence Exc 306

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Auteurs

Marcus Lettau (M)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany. Marcus.Lettau@uksh.de.

Michelle Dietz (M)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Sarah Vollmers (S)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Fred Armbrust (F)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Christian Peters (C)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Thi Mai Dang (TM)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Guranda Chitadze (G)

Medical Department II, Unit for Hematological Diagnostics, University Hospital Schleswig-Holstein, Langer Segen 8-10, 24105, Kiel, Germany.

Dieter Kabelitz (D)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

Ottmar Janssen (O)

Institute of Immunology, Christian-Albrechts University Kiel and University Hospital Schleswig-Holstein, Arnold-Heller-Str. 3, Bldg. 17, 24105, Kiel, Germany.

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Classifications MeSH