Interaction of nutritional status and diabetes on active and latent tuberculosis: a cross-sectional analysis.


Journal

BMC infectious diseases
ISSN: 1471-2334
Titre abrégé: BMC Infect Dis
Pays: England
ID NLM: 100968551

Informations de publication

Date de publication:
16 Jul 2019
Historique:
received: 09 03 2019
accepted: 30 06 2019
entrez: 18 7 2019
pubmed: 18 7 2019
medline: 18 10 2019
Statut: epublish

Résumé

Malnutrition and diabetes are risk factors for active tuberculosis (TB), possible risk factors for latent TB infection (LTBI), and may interact to alter their effect on these outcomes. Studies to date have not investigated this interaction. We enrolled 919 newly diagnosed active TB patients and 1113 household contacts at Primary Health Centres in Puducherry and Tamil Nadu, India from 2014 to 2018. In cross-sectional analyses, we used generalized estimating equations to measure additive and multiplicative interaction of body mass index (BMI) and diabetes on two outcomes, active TB and LTBI. Among overweight or obese adults, active TB prevalence was 12-times higher in diabetic compared to non-diabetic participants, 2.5-times higher among normal weight adults, and no different among underweight adults (P for interaction < 0.0001). Diabetes was associated with 50 additional active TB cases per 100 overweight or obese participants, 56 per 100 normal weight participants, and 17 per 100 underweight participants (P for interaction < 0.0001). Across BMI categories, screening 2.3-3.8 active TB patients yielded one hyperglycemic patient. LTBI prevalence did not differ by diabetes and BMI*diabetes interaction was not significant. BMI and diabetes are associated with newly diagnosed active TB, but not LTBI. Diabetes conferred the greatest risk of active TB in overweight and obese adults whereas the burden of active TB associated with diabetes was similar for normal and overweight or obese adults. Hyperglycemia was common among all active TB patients. These findings highlight the importance of bi-directional diabetes-active TB screening in India.

Sections du résumé

BACKGROUND BACKGROUND
Malnutrition and diabetes are risk factors for active tuberculosis (TB), possible risk factors for latent TB infection (LTBI), and may interact to alter their effect on these outcomes. Studies to date have not investigated this interaction.
METHODS METHODS
We enrolled 919 newly diagnosed active TB patients and 1113 household contacts at Primary Health Centres in Puducherry and Tamil Nadu, India from 2014 to 2018. In cross-sectional analyses, we used generalized estimating equations to measure additive and multiplicative interaction of body mass index (BMI) and diabetes on two outcomes, active TB and LTBI.
RESULTS RESULTS
Among overweight or obese adults, active TB prevalence was 12-times higher in diabetic compared to non-diabetic participants, 2.5-times higher among normal weight adults, and no different among underweight adults (P for interaction < 0.0001). Diabetes was associated with 50 additional active TB cases per 100 overweight or obese participants, 56 per 100 normal weight participants, and 17 per 100 underweight participants (P for interaction < 0.0001). Across BMI categories, screening 2.3-3.8 active TB patients yielded one hyperglycemic patient. LTBI prevalence did not differ by diabetes and BMI*diabetes interaction was not significant.
CONCLUSIONS CONCLUSIONS
BMI and diabetes are associated with newly diagnosed active TB, but not LTBI. Diabetes conferred the greatest risk of active TB in overweight and obese adults whereas the burden of active TB associated with diabetes was similar for normal and overweight or obese adults. Hyperglycemia was common among all active TB patients. These findings highlight the importance of bi-directional diabetes-active TB screening in India.

Identifiants

pubmed: 31311495
doi: 10.1186/s12879-019-4244-4
pii: 10.1186/s12879-019-4244-4
pmc: PMC6636094
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

627

Subventions

Organisme : NIDDK NIH HHS
ID : P30 DK017047
Pays : United States
Organisme : NCATS NIH HHS
ID : TL1 TR002318
Pays : United States
Organisme : CRDF Global
ID : USB1-31150-XX-13
Organisme : National Science Foundation
ID : OISE-9531011

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Auteurs

Rachel W Kubiak (RW)

Department of Epidemiology, University of Washington, 1959 NE Pacific Street, Health Sciences Building, Box 357236, Seattle, WA, 98195, USA. rwkubiak@uw.edu.

Sonali Sarkar (S)

Department of Preventive and Social Medicine, Jawaharlal Institute of Postgraduate Medical Education and Research, Pondicherry, India.

C Robert Horsburgh (CR)

Department of Epidemiology, Boston University School of Public Health, Boston, USA.
Section of Infectious Diseases, Department of Medicine, Boston University School of Medicine, Boston, USA.

Gautam Roy (G)

Department of Preventive and Social Medicine, Jawaharlal Institute of Postgraduate Medical Education and Research, Pondicherry, India.

Mario Kratz (M)

Department of Epidemiology, University of Washington, 1959 NE Pacific Street, Health Sciences Building, Box 357236, Seattle, WA, 98195, USA.
Division of Metabolism, Endocrinology, and Nutrition, Department of Medicine, University of Washington, Seattle, USA.
Cancer Prevention Program, Division of Public Health Sciences, Fred Hutchinson Cancer Research Center, Seattle, USA.

Ayiraveetil Reshma (A)

Department of Preventive and Social Medicine, Jawaharlal Institute of Postgraduate Medical Education and Research, Pondicherry, India.

Selby Knudsen (S)

Department of Epidemiology, Boston University School of Public Health, Boston, USA.

Padmini Salgame (P)

Center for Emerging Pathogens, Department of Medicine, Rutgers-New Jersey Medical School, Newark, USA.

Jerrold J Ellner (JJ)

Department of Epidemiology, Boston University School of Public Health, Boston, USA.
Center for Emerging Pathogens, Department of Medicine, Rutgers-New Jersey Medical School, Newark, USA.
Boston Medical Center, Boston, USA.

Paul K Drain (PK)

Department of Epidemiology, University of Washington, 1959 NE Pacific Street, Health Sciences Building, Box 357236, Seattle, WA, 98195, USA.
Department of Global Health, University of Washington, Seattle, USA.
Department of Medicine, University of Washington, Seattle, USA.

Natasha S Hochberg (NS)

Department of Epidemiology, Boston University School of Public Health, Boston, USA.
Section of Infectious Diseases, Department of Medicine, Boston University School of Medicine, Boston, USA.
Boston Medical Center, Boston, USA.

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