Loss of primary cilia increases polycystin-2 and TRPV4 and the appearance of a nonselective cation channel in the mouse cortical collecting duct.
Animals
Calcium Signaling
Cilia
/ metabolism
Disease Models, Animal
Female
Kidney Tubules, Collecting
/ metabolism
Male
Membrane Potentials
Mice, Knockout
Polycystic Kidney Diseases
/ genetics
TRPP Cation Channels
/ genetics
TRPV Cation Channels
/ genetics
Time Factors
Tumor Suppressor Proteins
/ deficiency
Up-Regulation
calcium channel
ciliopathy
mechanosensor
polycystic kidney disease
transient receptor potential
Journal
American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990
Informations de publication
Date de publication:
01 09 2019
01 09 2019
Historique:
pubmed:
18
7
2019
medline:
24
3
2020
entrez:
18
7
2019
Statut:
ppublish
Résumé
Flow-related bending of cilia results in Ca
Identifiants
pubmed: 31313950
doi: 10.1152/ajprenal.00210.2019
pmc: PMC6766628
doi:
Substances chimiques
TRPP Cation Channels
0
TRPV Cation Channels
0
Tg737Rpw protein, mouse
0
Trpv4 protein, mouse
0
Tumor Suppressor Proteins
0
polycystic kidney disease 1 protein
0
polycystic kidney disease 2 protein
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
F632-F637Subventions
Organisme : NIDDK NIH HHS
ID : K08 DK106465
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK110409
Pays : United States
Organisme : NIDDK NIH HHS
ID : R03 DK119717
Pays : United States
Organisme : NIDDK NIH HHS
ID : P30 DK074038
Pays : United States
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